Literature DB >> 21271611

IκB-Kinase-ε (IKKε/IKKi/IκBKε) expression and localization in prostate cancer tissues.

Benjamin Péant1, Valérie Forest, Vincent Trudeau, Mathieu Latour, Anne-Marie Mes-Masson, Fred Saad.   

Abstract

BACKGROUND: Advanced prostate cancer (PCa) remains a one of the leading causes of cancer related death and is often due to the progression from a hormone sensitive (HS) to a castrate resistant (CR) state for which therapeutic alternatives remain palliative. Molecular events involved in the progression to CR-PCa remain largely unknown. A previous study reported significantly higher levels of Iκ-B kinase-epsilon (IKKε) expression in CR compared to androgen-responsive cell lines. In the present study, we evaluate IKKε expression in human prostate tissue.
METHODS: In order to evaluate the modulation of IKKε expression in PCa tissue IKKε immunostaining was performed on paraffin-embedded prostate tissue microarrays containing cores from normal tissues (n = 47), non-malignant tissues adjacent to the tumor (n = 53), prostatic intraepithelial neoplasia (PIN) (n = 28), HS (n = 62), and CR tumors (n = 31).
RESULTS: We found a low cytoplasmic expression of IKKε in non-malignant tissue. HS tumors showed a significant increase in cytoplasmic IKKε expression compared to non-malignant tissues. CR tissues presented the highest cytoplasmic IKKε expression levels. We also report, for the first time, the presence of a nuclear localization of IKKε in prostate epithelial cells, in particular we observed an increase of IKKε nuclear localization in HS malignant tissues. Finally, we found a strong link between an increase of IKKε cytoplasmic expression in PCa and metastatic progression.
CONCLUSION: This study strongly suggests the role of IKKε as a PCa oncogene that may be involved in the emergence of a CR state.
Copyright © 2011 Wiley-Liss, Inc.

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Year:  2011        PMID: 21271611     DOI: 10.1002/pros.21329

Source DB:  PubMed          Journal:  Prostate        ISSN: 0270-4137            Impact factor:   4.104


  13 in total

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4.  Proteasome inhibition by bortezomib increases IL-8 expression in androgen-independent prostate cancer cells: the role of IKKα.

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Journal:  Nucleic Acids Res       Date:  2020-06-04       Impact factor: 16.971

8.  IKKi deficiency promotes pressure overload-induced cardiac hypertrophy and fibrosis.

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9.  IκB-Kinase-epsilon (IKKε) over-expression promotes the growth of prostate cancer through the C/EBP-β dependent activation of IL-6 gene expression.

Authors:  Benjamin Péant; Sophie Gilbert; Cécile Le Page; Alexis Poisson; Emilie L'Ecuyer; Zied Boudhraa; Marc Nicolas Bienz; Nathalie Delvoye; Fred Saad; Anne-Marie Mes-Masson
Journal:  Oncotarget       Date:  2017-02-28

10.  The breast cancer oncogene IKKε coordinates mitochondrial function and serine metabolism.

Authors:  Ruoyan Xu; William Jones; Ewa Wilcz-Villega; Ana Sh Costa; Vinothini Rajeeve; Robert B Bentham; Kevin Bryson; Ai Nagano; Busra Yaman; Sheila Olendo Barasa; Yewei Wang; Claude Chelala; Pedro Cutillas; Gyorgy Szabadkai; Christian Frezza; Katiuscia Bianchi
Journal:  EMBO Rep       Date:  2020-08-11       Impact factor: 8.807

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