Literature DB >> 21247853

Parathyroid carcinoma arising from four-gland hyperplasia.

Rondi M Kauffmann1, C Christofer Juhlin, Laurel E Fohn, James T Broome, John E Phay.   

Abstract

OBJECTIVE: To report the use of immunohistochemical staining for parafibromin, APC, and galectin-3 to evaluate the malignant potential of a resected parathyroid specimen in a patient initially presenting with primary hyperparathyroidism attributable to 4-gland hyperplasia, who subsequently developed metastatic parathyroid carcinoma.
METHODS: We describe a patient with primary hyperparathyroidism who underwent a 3-gland resection of hypercellular parathyroid glands, with postoperative normalization of her serum calcium and parathyroid hormone levels. She returned 4 years later with recurrent hypercalcemia and underwent partial resection of her remaining hypercellular parathyroid gland, without improvement of her hypercalcemia. Selective venous sampling localized the source as draining into her azygos vein, and metastatic parathyroid carcinoma was ultimately diagnosed.
RESULTS: Immunohistochemical staining for parafibromin, APC, and galectin-3 suggested the malignant potential of the atypical adenoma removed during the patient's original operation, which is believed to be the source of her metastatic disease. Access to this information by the treating surgeon may have prompted a more extensive en bloc resection or more vigilant follow-up that could have altered the patient's clinical course.
CONCLUSION: Immunohistochemical staining for parafibromin, APC, and galectin-3 can be used to help distinguish the source of metastatic disease in patients with parathyroid carcinoma. Selective venous sampling may help localize metastatic parathyroid carcinoma when the source is otherwise not apparent.

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Year:  2011        PMID: 21247853      PMCID: PMC3076624          DOI: 10.4158/EP10211.CR

Source DB:  PubMed          Journal:  Endocr Pract        ISSN: 1530-891X            Impact factor:   3.443


  18 in total

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2.  Loss of parafibromin expression in a subset of parathyroid adenomas.

Authors:  C Juhlin; C Larsson; T Yakoleva; I Leibiger; B Leibiger; A Alimov; G Weber; A Höög; A Villablanca
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Review 3.  Prognostic and predictive factors in endocrine tumours.

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4.  Loss of nuclear expression of parafibromin distinguishes parathyroid carcinomas and hyperparathyroidism-jaw tumor (HPT-JT) syndrome-related adenomas from sporadic parathyroid adenomas and hyperplasias.

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6.  Loss of expression for the Wnt pathway components adenomatous polyposis coli and glycogen synthase kinase 3-beta in parathyroid carcinomas.

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7.  Genomic localization of novel candidate tumor suppressor gene loci in human parathyroid adenomas.

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9.  Parafibromin immunoreactivity: its use as an additional diagnostic marker for parathyroid tumor classification.

Authors:  C C Juhlin; A Villablanca; K Sandelin; F Haglund; J Nordenström; L Forsberg; R Bränström; T Obara; A Arnold; C Larsson; A Höög
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10.  Loss of the retinoblastoma tumor-suppressor gene in parathyroid carcinoma.

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1.  Mortality factors in recurrent parathyroid cancer: a pooled analysis.

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  1 in total

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