Literature DB >> 21226546

Increased Th17 cells in coronary artery disease are associated with neutrophilic inflammation.

Zikuan Wang1, Jun Lee, Ye Zhang, Hongtao Wang, Xiongtao Liu, Fujun Shang, Qiangsun Zheng.   

Abstract

OBJECTIVE: Atherosclerosis is a progressive disease characterized by a series of inflammatory responses in the large and medium arteries. Th17 cells, a distinct T cell lineage which has recently been identified, have a proinflammatory role and are implicated in many inflammatory conditions in humans and mice. The present study was designed to assess whether Th17 cells are associated with human coronary atherosclerosis.
DESIGN: Flow cytometry was used to examine Th17 cell frequencies in patients with coronary atherosclerosis and in healthy individuals. ELISA and real-time RT-PCR were performed to investigate circulating interleukin (IL)-17 (the signature cytokine of Th17 cells) and IL-8 (the cytokine induced by IL-17) protein and mRNA levels.
RESULTS: Significantly increased Th17 cell frequencies are observed in patients with coronary artery disease compared to healthy controls. The protein and mRNA levels of IL-17 and IL-8 are also significantly elevated in patients with atherosclerosis compared to healthy volunteers. Furthermore, mRNA levels of IL-17 and IL-8 are correlated with each other and with peripheral neutrophil counts.
CONCLUSIONS: Our findings indicate that Th17 cells and their signature cytokine are involved in the process of atherogenesis. These data suggest that Th17 cells link T cell activity with neutrophilic inflammation in atherosclerosis.

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Year:  2011        PMID: 21226546     DOI: 10.3109/14017431.2010.491123

Source DB:  PubMed          Journal:  Scand Cardiovasc J        ISSN: 1401-7431            Impact factor:   1.589


  8 in total

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Review 2.  Current views on the functions of interleukin-17A-producing cells in atherosclerosis.

Authors:  M Butcher; E Galkina
Journal:  Thromb Haemost       Date:  2011-09-22       Impact factor: 5.249

3.  miR-19a mitigates hypoxia/reoxygenation-induced injury by depressing CCL20 and inactivating MAPK pathway in human embryonic cardiomyocytes.

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5.  PKM2-dependent metabolic skewing of hepatic Th17 cells regulates pathogenesis of non-alcoholic fatty liver disease.

Authors:  Maria E Moreno-Fernandez; Daniel A Giles; Jarren R Oates; Calvin C Chan; Michelle S M A Damen; Jessica R Doll; Traci E Stankiewicz; Xiaoting Chen; Kashish Chetal; Rebekah Karns; Matthew T Weirauch; Lindsey Romick-Rosendale; Stavra A Xanthakos; Rachel Sheridan; Sara Szabo; Amy S Shah; Michael A Helmrath; Thomas H Inge; Hitesh Deshmukh; Nathan Salomonis; Senad Divanovic
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Review 6.  The regulation of immune cells by Lactobacilli: a potential therapeutic target for anti-atherosclerosis therapy.

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7.  The Relationship between Epicardial Adipose Tissue Thickness and Serum Interleukin-17a Level in Patients with Isolated Metabolic Syndrome.

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8.  Increased peripheral proinflammatory T helper subsets contribute to cardiovascular complications in diabetic patients.

Authors:  Ru-xing Zhao; Wen-juan Li; Yi-ran Lu; Jun Qin; Chuan-long Wu; Meng Tian; Tian-yi He; Shou-nan Yi; Dong-qi Tang; Lei Sun; Li Chen
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  8 in total

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