Literature DB >> 21212396

Endogenous tissue plasminogen activator mediates bone marrow stromal cell-induced neurite remodeling after stroke in mice.

Li Hong Shen1, Hongqi Xin, Yi Li, Rui Lan Zhang, Yisheng Cui, Li Zhang, Mei Lu, Zheng Gang Zhang, Michael Chopp.   

Abstract

BACKGROUND AND
PURPOSE: Bone marrow stromal cells (BMSC) decrease neurological deficits in rodents after stroke and concomitantly induce extensive neurite remodeling in the brain, which highly correlates with the improvement of neurological function. We investigated the effects of endogenous tissue plasminogen activator (tPA) on neurite remodeling after BMSC treatment.
METHODS: Adult C57BL/6 wild-type (WT) mice and tPA knockout (tPA(-/-)) mice were subjected to middle cerebral artery occlusion, followed by an injection of 1×10(6) BMSC (n=18) or phosphate-buffered saline (n=18) into the tail vein 24 hours later. Behavioral tests were performed at 3, 7, and 14 days after middle cerebral artery occlusion. Animals were euthanized at 14 days after stroke.
RESULTS: The effects of BMSC on functional recovery depended on presence or absence of tPA, even after adjusting for imbalanced stroke severity. BMSC significantly improve functional recovery in WT mice compared to WT controls but show no beneficial effect in the tPA(-/-) mice compared to tPA(-/-) controls. Axonal density and synaptophysin-positive areas along the ischemic boundary zone of the cortex and striatum in WT mice are significantly higher than in the tPA(-/-) mice. BMSC treatment significantly increases tPA protein level and activity only in WT mice.
CONCLUSIONS: Our results suggest that endogenous tPA promotes BMSC-induced neurite outgrowth and may contribute to functional recovery after stroke.

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Year:  2011        PMID: 21212396      PMCID: PMC3093714          DOI: 10.1161/STROKEAHA.110.593863

Source DB:  PubMed          Journal:  Stroke        ISSN: 0039-2499            Impact factor:   7.914


  53 in total

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Authors:  Petti T Pang; Henry K Teng; Eugene Zaitsev; Newton T Woo; Kazuko Sakata; Shushuang Zhen; Kenneth K Teng; Wing-Ho Yung; Barbara L Hempstead; Bai Lu
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5.  Temporal profile of tissue plasminogen activator (tPA) and inhibitor expression after transient focal cerebral ischemia.

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Review 3.  The Neuroprotective Roles of Sonic Hedgehog Signaling Pathway in Ischemic Stroke.

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Review 4.  Cell based therapies for ischemic stroke: from basic science to bedside.

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7.  Multipotent mesenchymal stromal cells increase tPA expression and concomitantly decrease PAI-1 expression in astrocytes through the sonic hedgehog signaling pathway after stroke (in vitro study).

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8.  Intravascular stem cell transplantation for stroke.

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9.  Multipotent mesenchymal stromal cells decrease transforming growth factor β1 expression in microglia/macrophages and down-regulate plasminogen activator inhibitor 1 expression in astrocytes after stroke.

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10.  tPA regulates neurite outgrowth by phosphorylation of LRP5/6 in neural progenitor cells.

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