Literature DB >> 21174183

Brief review: anesthetic neurotoxicity in the elderly, cognitive dysfunction and Alzheimer's disease.

Edward A Bittner1, Yun Yue, Zhongcong Xie.   

Abstract

PURPOSE: Postoperative cognitive decline in the elderly has emerged as a major health concern. In addition, there is a growing interest in the potential relationship between general anesthetic exposure and the onset and progression of Alzheimer's disease (AD). The available evidence of a possible association between anesthesia, surgery, and long-term cognitive effects, including AD, deserves consideration. In this review, we summarize the evidence for anesthesia-induced neurotoxicity in the elderly, while highlighting the limitations of existing data, and we put the literature into perspective for the clinician. PRINCIPAL
FINDINGS: A growing body of evidence suggests that general anesthetics may be neurotoxic to both young and aging brains. Much of the evidence originates from in vitro and in vivo studies with cells, rodents, and nonhuman primates. Despite the animal data suggesting a relationship between anesthesia and neurotoxicity in the elderly, a definitive link remains elusive in humans.
CONCLUSIONS: The possible relation between anesthetic neurotoxicity, postoperative cognitive dysfunction, and AD remains elusive. It remains unclear whether postoperative cognitive decline in the elderly is related more to perioperative stress and related medical co-morbidities.

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Year:  2010        PMID: 21174183      PMCID: PMC4248669          DOI: 10.1007/s12630-010-9418-x

Source DB:  PubMed          Journal:  Can J Anaesth        ISSN: 0832-610X            Impact factor:   5.063


  67 in total

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4.  Prolonged cognitive dysfunction in survivors of critical illness.

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Journal:  Anaesthesia       Date:  2005-09       Impact factor: 6.955

5.  Coronary artery bypass grafting is not a risk factor for dementia or Alzheimer disease.

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6.  Assessment of the emergence of Alzheimer's disease following coronary artery bypass graft surgery or percutaneous transluminal coronary angioplasty.

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7.  Isoflurane neuroprotection in hypoxic hippocampal slice cultures involves increases in intracellular Ca2+ and mitogen-activated protein kinases.

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8.  Spatial memory performance 2 weeks after general anesthesia in adult rats.

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9.  Long-term postoperative cognitive dysfunction in the elderly ISPOCD1 study. ISPOCD investigators. International Study of Post-Operative Cognitive Dysfunction.

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  33 in total

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2.  The potential dual effects of anesthetic isoflurane on hypoxia-induced caspase-3 activation and increases in β-site amyloid precursor protein-cleaving enzyme levels.

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3.  2-Deoxy-D-glucose attenuates isoflurane-induced cytotoxicity in an in vitro cell culture model of H4 human neuroglioma cells.

Authors:  Jun Zhang; Yuanlin Dong; Zhipeng Xu; Yiying Zhang; Chuxiong Pan; Sayre McAuliffe; Fumito Ichinose; Yun Yue; Weimin Liang; Zhongcong Xie
Journal:  Anesth Analg       Date:  2011-09-29       Impact factor: 5.108

4.  Propofol protects against angiotensin II-induced mouse hippocampal HT22 cells apoptosis via inhibition of p66Shc mitochondrial translocation.

Authors:  Minmin Zhu; Jiawei Chen; Meilin Wen; Zhirong Sun; Xia Sun; Jing Wang; Changhong Miao
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5.  Anesthetics isoflurane and desflurane differently affect mitochondrial function, learning, and memory.

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6.  Effects of anesthetic isoflurane and desflurane on human cerebrospinal fluid Aβ and τ level.

Authors:  Bin Zhang; Ming Tian; Hui Zheng; Yu Zhen; Yun Yue; Tianzuo Li; Shuren Li; Edward R Marcantonio; Zhongcong Xie
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7.  Minocycline alleviates sevoflurane-induced cognitive impairment in aged rats.

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10.  Hippocampal volume reduction in elderly patients at risk for postoperative cognitive dysfunction.

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