Literature DB >> 21161698

Prostacyclin infusion may prevent secondary damage in pericontusional brain tissue.

Peter Reinstrup1, Carl-Henrik Nordström.   

Abstract

BACKGROUND: Prostacyclin is a potent vasodilator, inhibitor of leukocyte adhesion, and platelet aggregation, and has been suggested as therapy for cerebral ischemia. A case of focal traumatic brain lesion that was monitored using intracerebral microdialysis, and bedside analysis and display is reported here. When biochemical signs of cerebral ischemia progressed, i.v. infusion of prostacyclin was started.
METHODS: Two microdialysis catheters were placed in the penumbra zones surrounding evacuated focal brain contusions. The samples were analyzed for glucose, pyruvate, lactate, glutamate, and glycerol.
RESULTS: When biochemical deterioration indicated progressive secondary ischemia (increase in lactate/pyruvate ratio, decrease in glucose, and increase in glutamate levels), continuous infusion of prostacyclin (0.5-1.0 ng kg(-1) min(-1) i.v.) was started. The treatment resulted in an improvement of the lactate/pyruvate ratios and a normalization of the interstitial levels of glucose and glutamate. The glycerol levels remained within normal limits indicating that degradation of cellular membranes had not occurred.
CONCLUSION: The above case supports the view that new therapies directed toward protection of the sensitive biochemical penumbra zones surrounding focal brain lesions may be evaluated by intracerebral microdialysis.

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Year:  2011        PMID: 21161698     DOI: 10.1007/s12028-010-9486-3

Source DB:  PubMed          Journal:  Neurocrit Care        ISSN: 1541-6933            Impact factor:   3.210


  23 in total

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Journal:  J Cereb Blood Flow Metab       Date:  2007-01-17       Impact factor: 6.200

2.  An outcome study of severe traumatic head injury using the "Lund therapy" with low-dose prostacyclin.

Authors:  S Naredi; M Olivecrona; C Lindgren; A L Ostlund; P O Grände; L O Koskinen
Journal:  Acta Anaesthesiol Scand       Date:  2001-04       Impact factor: 2.105

3.  Interstitial glycerol as a marker for membrane phospholipid degradation in the acutely injured human brain.

Authors:  L Hillered; J Valtysson; P Enblad; L Persson
Journal:  J Neurol Neurosurg Psychiatry       Date:  1998-04       Impact factor: 10.154

Review 4.  Biology and therapeutic potential of prostacyclin.

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5.  Infusion of prostacyclin following experimental brain injury in the rat reduces cortical lesion volume.

Authors:  P Bentzer; G Mattiasson; T K McIntosh; T Wieloch; P O Grande
Journal:  J Neurotrauma       Date:  2001-03       Impact factor: 5.269

6.  Prostacyclin treatment normalises the MCA flow velocity in nimodipine-resistant cerebral vasospasm after aneurysmal subarachnoid haemorrhage: a pilot study.

Authors:  Lars-Owe D Koskinen; Magnus Olivecrona; Marie Rodling-Wahlström; Silvana Naredi
Journal:  Acta Neurochir (Wien)       Date:  2009-04-07       Impact factor: 2.216

Review 7.  Prostacyclin and analogues for acute ischaemic stroke.

Authors:  P M W Bath
Journal:  Cochrane Database Syst Rev       Date:  2004

8.  Prostacyclin treatment in severe traumatic brain injury: a microdialysis and outcome study.

Authors:  Magnus Olivecrona; Marie Rodling-Wahlström; Silvana Naredi; Lars-Owe D Koskinen
Journal:  J Neurotrauma       Date:  2009-08       Impact factor: 5.269

9.  Synaptic activity mediates death of hypoxic neurons.

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10.  Low-dose prostacyclin improves cortical perfusion following experimental brain injury in the rat.

Authors:  Peter Bentzer; Daniele Venturoli; Ola Carlsson; P-O Grände
Journal:  J Neurotrauma       Date:  2003-05       Impact factor: 5.269

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2.  Bedside diagnosis of mitochondrial dysfunction after malignant middle cerebral artery infarction.

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4.  The effects of continuous prostacyclin infusion on regional blood flow and cerebral vasospasm following subarachnoid haemorrhage: study protocol for a randomised controlled trial.

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Review 5.  Aspects on the Physiological and Biochemical Foundations of Neurocritical Care.

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Review 6.  Critical Evaluation of the Lund Concept for Treatment of Severe Traumatic Head Injury, 25 Years after Its Introduction.

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