Literature DB >> 21159048

ZM447439, the Aurora kinase B inhibitor, suppresses the growth of cervical cancer SiHa cells and enhances the chemosensitivity to cisplatin.

Liya Zhang1, Shulan Zhang.   

Abstract

AIM: To investigate the effects of an Aurora kinase B inhibitor (ZM447439) on the cervical cancer cell line SiHa and chemotherapy of cisplatin (cDDP). MATERIALS &
METHODS: Detected Aurora-B protein in different tissues of the cervix by immunohistochemistry and then analyzed the relationship between Aurora B protein and clinical parameters of cervical cancer. The effect and synergistic effect of ZM447439 and cDDP on proliferation of SiHa cells was tested by MTT. The changes of cell cycle and apoptosis were detected by flow cytometry. Aurora-B, histone H3 phosphorylation (H3-P) protein, human papillomavirus16 E6 (HPV16E6) and BCL-2, P53, VEGF protein were detected by Western blot.
RESULTS: The positive rate of Aurora-B expression was the highest in cervical cancer and had no significant correlation with clinical stage, lymph node metastasis and age. ZM447439 can reduce the number of SiHa cells, increase the volume of cells and lead to apoptosis. The growth of SiHa cells treated with ZM447439, cDDP and the combination of both was inhibited in dose- and time-dependent manners. The inhibition rate of the combined treatment is significantly higher than that of two other single drug groups (P < 0.05). The synergistic effect is observed in the combined therapy. S-phase arrest and early apoptosis has become more evident in the combined treatment. ZM447439 significantly inhibited the expression of Aurora-B and H3-P protein (P < 0.05). ZM447439, cDDP and the combination of both reduced the expression of HPV16E6 and BCL-2 protein, raised P53 protein expression (P < 0.05), whose effects were more obvious in the combined therapy. cDDP could also reduce VEGF protein expression, but ZM447439 could not.
CONCLUSION: Our results suggest that Aurora-B may represent a valid target in cervical squamous carcinoma and has a synergistic effect with cDDP.
© 2010 The Authors. Journal of Obstetrics and Gynaecology Research © 2010 Japan Society of Obstetrics and Gynecology.

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Year:  2010        PMID: 21159048     DOI: 10.1111/j.1447-0756.2010.01414.x

Source DB:  PubMed          Journal:  J Obstet Gynaecol Res        ISSN: 1341-8076            Impact factor:   1.730


  12 in total

1.  Inhibition of Aurora kinases enhances chemosensitivity to temozolomide and causes radiosensitization in glioblastoma cells.

Authors:  Kleiton Silva Borges; Angel Maurício Castro-Gamero; Daniel Antunes Moreno; Vanessa da Silva Silveira; Maria Sol Brassesco; Rosane Gomes de Paula Queiroz; Harley Francisco de Oliveira; Carlos Gilberto Carlotti; Carlos Alberto Scrideli; Luiz Gonzaga Tone
Journal:  J Cancer Res Clin Oncol       Date:  2011-12-09       Impact factor: 4.553

2.  Combined inhibition of cellular pathways as a future therapeutic option in fatal anaplastic thyroid cancer.

Authors:  Annette Wunderlich; Silvia Roth; Annette Ramaswamy; Brandon H Greene; Cornelia Brendel; Ulrike Hinterseher; Detlef K Bartsch; Sebastian Hoffmann
Journal:  Endocrine       Date:  2012-04-05       Impact factor: 3.633

3.  Analysis and Modeling of Chromosome Congression During Mitosis in the Chemotherapy Drug Cisplatin.

Authors:  Jeremy M Chacón; Melissa K Gardner
Journal:  Cell Mol Bioeng       Date:  2013-12-01       Impact factor: 2.321

4.  In vitro evaluation of a combination treatment involving anticancer agents and an aurora kinase B inhibitor.

Authors:  Senna Sakai; Hiroto Izumi; Yukiko Yoshiura; Yoshifumi Nakayama; Takahiro Yamaguchi; Yoshikazu Harada; Chiho Koi; Hiroyuki Kurata; Yasuo Morimoto
Journal:  Oncol Lett       Date:  2016-09-20       Impact factor: 2.967

5.  Network analysis revealed aurora kinase dysregulation in five gynecological types of cancer.

Authors:  Shikha Suman; Ashutosh Mishra
Journal:  Oncol Lett       Date:  2017-11-08       Impact factor: 2.967

6.  Sequential treatment with aurora B inhibitors enhances cisplatin-mediated apoptosis via c-Myc.

Authors:  Yaxi Ma; Handi Cao; Siyue Lou; Xuejing Shao; Wen Lv; Xiaotian Qi; Yujia Liu; Meidan Ying; Qiaojun He; Xiaochun Yang
Journal:  J Mol Med (Berl)       Date:  2014-11-21       Impact factor: 4.599

Review 7.  The Aurora kinase inhibitors in cancer research and therapy.

Authors:  Jonas Cicenas
Journal:  J Cancer Res Clin Oncol       Date:  2016-03-01       Impact factor: 4.553

Review 8.  Aurora kinase inhibitors: Potential molecular-targeted drugs for gynecologic malignant tumors.

Authors:  Kiyoko Umene; Kouji Banno; Iori Kisu; Megumi Yanokura; Yuya Nogami; Kosuke Tsuji; Kenta Masuda; Arisa Ueki; Yusuke Kobayashi; Wataru Yamagami; Hiroyuki Nomura; Eiichiro Tominaga; Nobuyuki Susumu; Daisuke Aoki
Journal:  Biomed Rep       Date:  2013-03-27

9.  Functional impact of Aurora A-mediated phosphorylation of HP1γ at serine 83 during cell cycle progression.

Authors:  Adrienne Grzenda; Phoebe Leonard; Seungmae Seo; Angela J Mathison; Guillermo Urrutia; Ezequiel Calvo; Juan Iovanna; Raul Urrutia; Gwen Lomberk
Journal:  Epigenetics Chromatin       Date:  2013-07-05       Impact factor: 4.954

10.  Reversine suppresses oral squamous cell carcinoma via cell cycle arrest and concomitantly apoptosis and autophagy.

Authors:  Ying-Ray Lee; Wei-Ching Wu; Wen-Tsai Ji; Jeff Yi-Fu Chen; Ya-Ping Cheng; Ming-Ko Chiang; Hau-Ren Chen
Journal:  J Biomed Sci       Date:  2012-01-27       Impact factor: 8.410

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