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Literature DB >> 2112092

Concurrence of transient asplenia and pure red cell aplasia.

Abstract

We report a 9-year-old male with anatomical asplenia diagnosed at 7 months of age documented by ultrasound and liver-spleen scan which resolved spontaneously 3 years later. The patient concurrently had pure red cell aplasia which subsequently resolved spontaneously.

Entities: Disease Species

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Year: 1990 PMID： 2112092 DOI： 10.1007/bf01957688

Source DB: PubMed Journal: Eur J Pediatr ISSN： 0340-6199 Impact factor: 3.183

10 in total

1. Implications of agenesis of the spleen on the pathogenesis of conotruncus anomalies in childhood; an analysis of the heart malformations in the splenic agenesis syndrome, with fourteen new cases.

Authors: B I IVEMARK
Journal: Acta Paediatr Suppl Date: 1955-11

2. Persistent B19 parvovirus infection as a cause of severe chronic anaemia in children with acute lymphocytic leukaemia.

Authors: G J Kurtzman; B Cohen; P Meyers; A Amunullah; N S Young
Journal: Lancet Date: 1988-11-19 Impact factor: 79.321

3. Pure red-cell aplasia of 10 years' duration due to persistent parvovirus B19 infection and its cure with immunoglobulin therapy.

Authors: G Kurtzman; N Frickhofen; J Kimball; D W Jenkins; A W Nienhuis; N S Young
Journal: N Engl J Med Date: 1989-08-24 Impact factor: 91.245

1 in total

1. Delayed splenic development or transient asplenia?

Authors: S Ozsoylu
Journal: Eur J Pediatr Date: 1991-04 Impact factor: 3.183

1 in total

Concurrence of transient asplenia and pure red cell aplasia.

1. Implications of agenesis of the spleen on the pathogenesis of conotruncus anomalies in childhood; an analysis of the heart malformations in the splenic agenesis syndrome, with fourteen new cases.

2. Persistent B19 parvovirus infection as a cause of severe chronic anaemia in children with acute lymphocytic leukaemia.

3. Pure red-cell aplasia of 10 years' duration due to persistent parvovirus B19 infection and its cure with immunoglobulin therapy.

4. Chronic bone marrow failure due to persistent B19 parvovirus infection.

5. Functional asplenia in sickle-cell anemia.

6. Parvovirus infections and hypoplastic crisis in sickle-cell anaemia.

7. Functional hyposplenia in cyanotic congenital heart disease.

Review 8. Hematologic and hematopoietic consequences of B19 parvovirus infection.

9. Congenital hypoplastic anemia (CHA) associated with congenital absence of the spleen.

10. Causes and temporal sequence of onset of functional asplenia in adults.

1. Delayed splenic development or transient asplenia?