Literature DB >> 21116834

Aspirin significantly decreases the nonopsonic phagocytosis and immunogenicity of macrophages in mice.

Aqeel Javeed1, Yuzhu Hou, Kaizhong Duan, Baojun Zhang, Hong Shen, Yuhong Cao, Yong Zhao.   

Abstract

OBJECTIVE AND
DESIGN: The effect of acetylsalicyclic acid (ASA) on the phagocytosis and immunogenicity of macrophages remains to be determined. MATERIAL OR
SUBJECTS: BALB/c mice and peritoneal macrophages were used. TREATMENT: BALB/c mice were treated with ASA (0, 6, or 60 mg/kg/day) intraperitoneally for 1 week.
METHODS: Flow cytometry and phagocytosis assay were employed.
RESULTS: ASA significantly decreased cell numbers, nonopsonic phagocytosis, and immunogenicity as well as changing the phenotypes of peritoneal macrophages in vivo. After in vitro treatment with ASA, macrophages expressed low levels of MHC-II, CD80, and CD47 molecules, and showed significantly decreased phagocytosis. Importantly, ASA blocked the differentiation of macrophages from bone marrow cells in vitro as indicated by decreased macrophage cell numbers and phenotype alteration.
CONCLUSIONS: The present study provides basic information on the direct effect of ASA on macrophages, supporting the potential application of ASA in patients with allo-grafts or autoimmune diseases.

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Year:  2010        PMID: 21116834     DOI: 10.1007/s00011-010-0283-4

Source DB:  PubMed          Journal:  Inflamm Res        ISSN: 1023-3830            Impact factor:   4.575


  34 in total

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