Literature DB >> 21113720

Evaluation of two commercialised in situ hybridisation assays for detecting HPV-DNA in formalin-fixed, paraffin-embedded tissue.

Maresa Montag1, Thomas J F Blankenstein, Naim Shabani, Ansgar Brüning, Ioannis Mylonas.   

Abstract

INTRODUCTION: The role of human papilloma virus (HPV) in the pathogenesis of anogenital dysplasia is now conclusive. However, HPV detection in formalin-fixed and paraffin-embedded tissues remains controversial. Therefore, the aim of this study was to evaluate morphological changes directly in tissue specimens using a HPV-DNA detection system involving HPV in situ hybridisation.
MATERIALS AND METHODS: Samples from patients with cervical carcinoma were analysed using the GenPoint HPV DNA Probe Cocktail (Dako, Glostrup, Denmark) and the ZytoFast HPV Screening CISH-Kit (Zytomed, Berlin, Germany). Three cervical carcinoma cell lines with a well-defined HPV copy number per cell (SiHa, HeLa, and CaSki) served as positive controls for sensitivity testing, while two HPV-negative cell lines (AC-1M32, MCF-7) and brain tissue samples served as negative controls. Moreover, to assess the validity of the in situ hybridisation, the expression of HPV-16 DNA in cell lines was demonstrated by HPV-16 E6-specific PCR.
RESULTS: Both HPV-screening assays revealed strong signals of episomal and integrated HPV-DNA at a HPV copy number of more than 50 copies/cell. All cervical carcinoma samples were positive in the Dako assay, which identifies 13 high-risk HPV genotypes, whereas HPV-DNA could be detected in 9/10 cervical carcinoma samples using the Zytofast assay, identifying HPV 16, 18, 31, 33, and 35.
CONCLUSION: HPV in situ hybridisation is a convenient and powerful tool for detecting HPV-DNA in formalin-fixed and paraffin-embedded tissue samples. Therefore, this technique is suitable for analysis of a potential HPV infection using archival pathological slides.

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Year:  2010        PMID: 21113720     DOI: 10.1007/s00404-010-1771-z

Source DB:  PubMed          Journal:  Arch Gynecol Obstet        ISSN: 0932-0067            Impact factor:   2.344


  2 in total

1.  Risk of progression of early cervical lesions is associated with integration and persistence of HPV-16 and expression of E6, Ki-67, and telomerase.

Authors:  Arianna Vega-Peña; Berenice Illades-Aguiar; Eugenia Flores-Alfaro; Esther López-Bayghen; Marco Antonio Leyva-Vázquez; Eduardo Castañeda-Saucedo; Luz Del Carmen Alarcón-Romero
Journal:  J Cytol       Date:  2013-10       Impact factor: 1.000

2.  Chromogenic in situ hybridization and p16/Ki67 dual staining on formalin-fixed paraffin-embedded cervical specimens: correlation with HPV-DNA test, E6/E7 mRNA test, and potential clinical applications.

Authors:  Roberta Zappacosta; Antonella Colasante; Patrizia Viola; Tommaso D'Antuono; Giuseppe Lattanzio; Serena Capanna; Daniela Maria Pia Gatta; Sandra Rosini
Journal:  Biomed Res Int       Date:  2013-11-24       Impact factor: 3.411

  2 in total

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