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Literature DB >> 21108388

Genetic drift. "Omics"as the filtering gateway between environment and phenotype: The inflammatory bowel diseases example.

Richard Kellermayer¹.

Abstract

Entities: Disease

Mesh：

Year: 2010 PMID： 21108388 DOI： 10.1002/ajmg.a.33726

Source DB: PubMed Journal: Am J Med Genet A ISSN： 1552-4825 Impact factor: 2.802

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Cited

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12 in total

1. Loss of n-6 fatty acid induced pediatric obesity protects against acute murine colitis.

Authors: Dorottya Nagy-Szakal; Sabina A V Mir; R Alan Harris; Scot E Dowd; Takeshi Yamada; H Daniel Lacorazza; Nina Tatevian; C Wayne Smith; Edwin F de Zoeten; John Klein; Richard Kellermayer
Journal: FASEB J Date: 2015-04-22 Impact factor: 5.191

2. The remarkable capacity for gut microbial and host interactions.

Authors: Dorottya Nagy-Szakal; Richard Kellermayer
Journal: Gut Microbes Date: 2011-05-01

3. Maternal micronutrients can modify colonic mucosal microbiota maturation in murine offspring.

Authors: Dorottya Nagy-Szakal; Matthew C Ross; Scot E Dowd; Sabina A V Mir; Tiffany D Schaible; Joseph F Petrosino; Richard Kellermayer
Journal: Gut Microbes Date: 2012-06-20

4. Genome-wide peripheral blood leukocyte DNA methylation microarrays identified a single association with inflammatory bowel diseases.

Authors: R Alan Harris; Dorottya Nagy-Szakal; Natalia Pedersen; Antone Opekun; Jiri Bronsky; Pia Munkholm; Cathrine Jespersgaard; PaalSkytt Andersen; Bela Melegh; George Ferry; Tine Jess; Richard Kellermayer
Journal: Inflamm Bowel Dis Date: 2012-03-29 Impact factor: 5.325

5. Maternal methyl-donor supplementation induces prolonged murine offspring colitis susceptibility in association with mucosal epigenetic and microbiomic changes.

Authors: Tiffany D Schaible; R Alan Harris; Scot E Dowd; C Wayne Smith; Richard Kellermayer
Journal: Hum Mol Genet Date: 2011-02-04 Impact factor: 6.150

Genetic drift. "Omics"as the filtering gateway between environment and phenotype: The inflammatory bowel diseases example.

1. Loss of n-6 fatty acid induced pediatric obesity protects against acute murine colitis.

2. The remarkable capacity for gut microbial and host interactions.

3. Maternal micronutrients can modify colonic mucosal microbiota maturation in murine offspring.

4. Genome-wide peripheral blood leukocyte DNA methylation microarrays identified a single association with inflammatory bowel diseases.

5. Maternal methyl-donor supplementation induces prolonged murine offspring colitis susceptibility in association with mucosal epigenetic and microbiomic changes.

6. SMAD4 haploinsufficiency associates with augmented colonic inflammation in select humans and mice.

Review 7. Epigenetics and the developmental origins of inflammatory bowel diseases.

Review 8. Microbiome-Epigenome Interactions and the Environmental Origins of Inflammatory Bowel Diseases.

Review 9. Ulcerative colitis as a polymicrobial infection characterized by sustained broken mucus barrier.

10. The C57BL/6J mouse exhibits sporadic congenital portosystemic shunts.