Literature DB >> 21097816

Effects of exercise on mitochondrial DNA content in skeletal muscle of patients with COPD.

Luis Puente-Maestu1, Alberto Lázaro, Alberto Tejedor, Sonia Camaño, Marta Fuentes, Miguel Cuervo, Beatriz Oláiz Navarro, Alvar Agustí.   

Abstract

BACKGROUND: Exhausting exercise reduces the mitochondrial DNA (mtDNA) content in the skeletal muscle of healthy subjects due to oxidative damage. Since patients with chronic obstructive pulmonary disease (COPD) suffer enhanced oxidative stress during exercise, it was hypothesised that the mtDNA content will be further reduced.
OBJECTIVE: To investigate the effects of exercise above and below the lactate threshold (LT) on the mtDNA content of skeletal muscle of patients with COPD.
METHODS: Eleven patients with COPD (67 ± 8 years; forced expiratory volume in 1 s (FEV1) 45 ± 8%ref) and 10 healthy controls (66 ± 4 years; FEV1 90 ± 7% ref) cycled 45 min above LT (65% peak oxygen uptake (V′o2peak) and another 7 patients (65 ± 6 years; FEV1 50 ± 4%ref) and 7 controls (56 ± 9 years; FEV1 92 ± 6% ref) cycled 45 min below their LT (50% V′o2peak). Biopsies from the vastus lateralis muscle were obtained before exercise, immediately after and 1 h, 1 day and 1 week later to determine by PCR the mtDNA/nuclear DNA (nDNA) ratio (a marker of mtDNA content) and the expression of the peroxisome proliferator-activated receptor-γcoactivator-1α (PGC-1α) mRNA and the amount of reactive oxygen species produced during exercise was estimated from total V′o2.
RESULTS: Skeletal muscle mtDNA/nDNA fell significantly after exercise above the LT both in controls and in patients with COPD, but the changes were greater in those with COPD. These changes correlated with production of reactive oxygen species, increases in manganese superoxide dismutase and PGC-1α mRNA and returned to baseline values 1 week later. This pattern of response was also observed, albeit minimised, in patients exercising below the LT.
CONCLUSIONS: In patients with COPD, exercise enhances the decrease in mtDNA content of skeletal muscle and the expression of PGC-1α mRNA seen in healthy subjects, probably due to oxidative stress.

Entities:  

Mesh:

Substances:

Year:  2010        PMID: 21097816     DOI: 10.1136/thx.2010.153031

Source DB:  PubMed          Journal:  Thorax        ISSN: 0040-6376            Impact factor:   9.139


  27 in total

1.  Quadriceps exercise intolerance in patients with chronic obstructive pulmonary disease: the potential role of altered skeletal muscle mitochondrial respiration.

Authors:  Jayson R Gifford; Joel D Trinity; Gwenael Layec; Ryan S Garten; Song-Young Park; Matthew J Rossman; Steen Larsen; Flemming Dela; Russell S Richardson
Journal:  J Appl Physiol (1985)       Date:  2015-08-13

Review 2.  Molecular and biological pathways of skeletal muscle dysfunction in chronic obstructive pulmonary disease.

Authors:  Esther Barreiro; Joaquim Gea
Journal:  Chron Respir Dis       Date:  2016-04-06       Impact factor: 2.444

3.  Relationship between PPARα mRNA expression and mitochondrial respiratory function and ultrastructure of the skeletal muscle of patients with COPD.

Authors:  Jian-Qing Zhang; Xiang-Yu Long; Yu Xie; Zhi-Huan Zhao; Li-Zhou Fang; Ling Liu; Wei-Ping Fu; Jing-Kui Shu; Jiang-Hai Wu; Lu-Ming Dai
Journal:  Bioengineered       Date:  2017-08-11       Impact factor: 3.269

Review 4.  Mitochondrial dysfunction and damage associated molecular patterns (DAMPs) in chronic inflammatory diseases.

Authors:  Charles S Dela Cruz; Min-Jong Kang
Journal:  Mitochondrion       Date:  2017-12-06       Impact factor: 4.160

Review 5.  Can muscle protein metabolism be specifically targeted by exercise training in COPD?

Authors:  Davina C M Simoes; Ioannis Vogiatzis
Journal:  J Thorac Dis       Date:  2018-05       Impact factor: 2.895

Review 6.  Muscle atrophy in chronic obstructive pulmonary disease: molecular basis and potential therapeutic targets.

Authors:  Esther Barreiro; Ariel Jaitovich
Journal:  J Thorac Dis       Date:  2018-05       Impact factor: 2.895

Review 7.  An official American Thoracic Society/European Respiratory Society statement: update on limb muscle dysfunction in chronic obstructive pulmonary disease.

Authors:  François Maltais; Marc Decramer; Richard Casaburi; Esther Barreiro; Yan Burelle; Richard Debigaré; P N Richard Dekhuijzen; Frits Franssen; Ghislaine Gayan-Ramirez; Joaquim Gea; Harry R Gosker; Rik Gosselink; Maurice Hayot; Sabah N A Hussain; Wim Janssens; Micheal I Polkey; Josep Roca; Didier Saey; Annemie M W J Schols; Martijn A Spruit; Michael Steiner; Tanja Taivassalo; Thierry Troosters; Ioannis Vogiatzis; Peter D Wagner
Journal:  Am J Respir Crit Care Med       Date:  2014-05-01       Impact factor: 21.405

8.  Altered skeletal muscle mitochondrial phenotype in COPD: disease vs. disuse.

Authors:  Jayson R Gifford; Joel D Trinity; Oh-Sung Kwon; Gwenael Layec; Ryan S Garten; Song-Young Park; Ashley D Nelson; Russell S Richardson
Journal:  J Appl Physiol (1985)       Date:  2017-12-28

9.  The Acute Effects of Exercise and Temperature on Regional mtDNA.

Authors:  Mark L McGlynn; Halee Schnitzler; Robert Shute; Brent Ruby; Dustin Slivka
Journal:  Int J Environ Res Public Health       Date:  2021-06-12       Impact factor: 3.390

10.  Suppression of mitochondrial electron transport chain function in the hypoxic human placenta: a role for miRNA-210 and protein synthesis inhibition.

Authors:  Francesca Colleoni; Nisha Padmanabhan; Hong-Wa Yung; Erica D Watson; Irene Cetin; Martha C Tissot van Patot; Graham J Burton; Andrew J Murray
Journal:  PLoS One       Date:  2013-01-30       Impact factor: 3.240

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.