Literature DB >> 21093310

Fluctuating and constant valproate administration gives equivalent seizure control in rats with genetic and acquired epilepsy.

S Dedeurwaerdere1, L van Raay, M J Morris, R C Reed, R E Hogan, T J O'Brien.   

Abstract

PURPOSE: Controlled-release formulations of Valproate (VPA) reduce side effects by minimizing peak plasma VPA concentrations in patients with epilepsy. However, the impact of this on anti-seizure efficacy has not been thoroughly explored. Here the pharmacokinetics and pharmacodynamics of chronic intermittent (consequently, peak VPA concentrations) and continuous VPA administration were directly compared in two rat models of epilepsy.
METHODS: Genetic Absence Epilepsy Rats from Strasbourg (GAERS) received a single acute bolus of VPA (100 mg/kg intravenously) combined with electroencephalography (EEG) and/or blood sampling for 180 min post-injection. GAERS and epileptic rats post-kainic acid-induced status epilepticus were chronically infused intravenously (3-5 days, respectively) with (i) saline followed by in random order (ii) intermittent and (iii) continuous VPA (42 mg/kg/h), separated by two days of wash-out. Seizures were quantified using video-EEG monitoring and VPA levels measured in brain, cerebrospinal fluid and plasma.
RESULTS: Following acute VPA administration seizure suppression in GAERS persisted after plasma VPA levels became very low. Chronic intermittent and continuous VPA significantly suppressed seizures in both models (p<0.01) with no difference between administration regimens. In GAERS, the pattern of seizure suppression during intermittent treatment was constant, in contrast to the fluctuating VPA plasma and brain levels. There was discordance between the temporal pattern of plasma, brain VPA levels and seizure suppression efficacy in GAERS.
CONCLUSION: Administration regimes that result in fluctuating VPA blood levels achieve equivalent sustained seizure suppression as those that maintain steady mid-range concentrations. Copyright Â
© 2010 British Epilepsy Association. Published by Elsevier Ltd. All rights reserved.

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Year:  2010        PMID: 21093310     DOI: 10.1016/j.seizure.2010.10.011

Source DB:  PubMed          Journal:  Seizure        ISSN: 1059-1311            Impact factor:   3.184


  3 in total

1.  Pronounced antiepileptic activity of the subtype-selective GABAA -positive allosteric modulator PF-06372865 in the GAERS absence epilepsy model.

Authors:  Venceslas Duveau; Derek L Buhl; Alexis Evrard; Céline Ruggiero; Betty Mandé-Niedergang; Corinne Roucard; Rachel Gurrell
Journal:  CNS Neurosci Ther       Date:  2018-08-12       Impact factor: 5.243

2.  Long-term valproate treatment increases brain neuropeptide Y expression and decreases seizure expression in a genetic rat model of absence epilepsy.

Authors:  Johanna Elms; Kim L Powell; Leena van Raay; Stefanie Dedeurwaerdere; Terence J O'Brien; Margaret J Morris
Journal:  PLoS One       Date:  2013-09-09       Impact factor: 3.240

3.  PET imaging of brain inflammation during early epileptogenesis in a rat model of temporal lobe epilepsy.

Authors:  Stefanie Dedeurwaerdere; Paul D Callaghan; Tien Pham; Gita L Rahardjo; Halima Amhaoul; Paula Berghofer; Mitchell Quinlivan; Filomena Mattner; Christian Loc'h; Andrew Katsifis; Marie-Claude Grégoire
Journal:  EJNMMI Res       Date:  2012-11-08       Impact factor: 3.138

  3 in total

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