Literature DB >> 20975569

Therapeutic goals in the treatment of Fabry disease.

Atul Mehta1, Michael L West, Guillem Pintos-Morell, Ricardo Reisin, Kathy Nicholls, Luis E Figuera, Rossella Parini, Luiz R Carvalho, Christoph Kampmann, Gregory M Pastores, Olivier Lidove.   

Abstract

PURPOSE: Fabry disease is a progressive multiorgan, multisystem disorder that is caused by a deficiency in the lysosomal enzyme α-galactosidase A. Serious renal, cardiac, and cerebrovascular involvement are responsible for much of the morbidity and premature mortality associated with Fabry disease, and neuropathic pain, gastrointestinal problems, and hypohidrosis negatively affect quality of life of patients with Fabry disease. Fabry disease is X-linked, but women are often symptomatic and may be as severely affected as men.
METHODS: We propose a series of therapeutic and symptomatic goals for use in setting the expectations of enzyme replacement therapy and for assessing the response to enzyme replacement therapy in the treatment of Fabry disease.
RESULTS: Enzyme replacement therapy has been available since 2001 and has been associated with benefit in clinical trials, including stabilization of kidney function, improvement of cardiac structure and function, reduction in severity of neuropathic pain, and improvement in gastrointestinal involvement.
CONCLUSIONS: The presentation of these therapeutic goals will aid in the evaluation of response to enzyme replacement therapy and be useful in establishing an overall management plan for individual patients.

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Year:  2010        PMID: 20975569     DOI: 10.1097/GIM.0b013e3181f6e676

Source DB:  PubMed          Journal:  Genet Med        ISSN: 1098-3600            Impact factor:   8.822


  8 in total

1.  Effects of switching from agalsidase Beta to agalsidase alfa in 10 patients with anderson-fabry disease.

Authors:  A Pisani; L Spinelli; B Visciano; I Capuano; M Sabbatini; E Riccio; G Messalli; M Imbriaco
Journal:  JIMD Rep       Date:  2012-10-21

Review 2.  Disease registries and outcomes research in children: focus on lysosomal storage disorders.

Authors:  Simon Jones; Emma James; Suyash Prasad
Journal:  Paediatr Drugs       Date:  2011-02-01       Impact factor: 3.022

3.  Renal outcomes of agalsidase beta treatment for Fabry disease: role of proteinuria and timing of treatment initiation.

Authors:  David G Warnock; Alberto Ortiz; Michael Mauer; Gabor E Linthorst; João P Oliveira; Andreas L Serra; László Maródi; Renzo Mignani; Bojan Vujkovac; Dana Beitner-Johnson; Roberta Lemay; J Alexander Cole; Einar Svarstad; Stephen Waldek; Dominique P Germain; Christoph Wanner
Journal:  Nephrol Dial Transplant       Date:  2011-07-29       Impact factor: 5.992

4.  An open-label clinical trial of agalsidase alfa enzyme replacement therapy in children with Fabry disease who are naïve to enzyme replacement therapy.

Authors:  Ozlem Goker-Alpan; Nicola Longo; Marie McDonald; Suma P Shankar; Raphael Schiffmann; Peter Chang; Yinghua Shen; Arian Pano
Journal:  Drug Des Devel Ther       Date:  2016-05-25       Impact factor: 4.162

5.  Correlations between Endomyocardial Biopsies and Cardiac Manifestations in Taiwanese Patients with the Chinese Hotspot IVS4+919G>A Mutation: Data from the Fabry Outcome Survey.

Authors:  Ting-Rong Hsu; Fu-Pang Chang; Tzu-Hung Chu; Shih-Hsien Sung; Svetlana Bizjajeva; Wen-Chung Yu; Dau-Ming Niu
Journal:  Int J Mol Sci       Date:  2017-01-09       Impact factor: 5.923

6.  Effectiveness of agalsidase alfa enzyme replacement in Fabry disease: cardiac outcomes after 10 years' treatment.

Authors:  Christoph Kampmann; Amandine Perrin; Michael Beck
Journal:  Orphanet J Rare Dis       Date:  2015-09-29       Impact factor: 4.123

Review 7.  Fabry nephropathy: a review - how can we optimize the management of Fabry nephropathy?

Authors:  Stephen Waldek; Sandro Feriozzi
Journal:  BMC Nephrol       Date:  2014-05-06       Impact factor: 2.388

8.  Clinical course of patients with Fabry disease who were switched from agalsidase-β to agalsidase-α.

Authors:  Kazuya Tsuboi; Hiroshi Yamamoto
Journal:  Genet Med       Date:  2014-03-20       Impact factor: 8.822

  8 in total

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