Literature DB >> 20871222

Clinical and biological significance of E-cadherin protein expression in invasive lobular carcinoma of the breast.

Emad A Rakha1, Arjun Patel, Des G Powe, Ahmed Benhasouna, Andrew R Green, Maryou B Lambros, Jorge S Reis-Filho, Ian O Ellis.   

Abstract

BACKGROUND: Although virtually all cases of lobular carcinoma in situ lack E-cadherin expression, a proportion of morphologically typical invasive lobular carcinomas (ILCs) retain its expression. The frequency and significance of E-cadherin expression in ILC remain to be elucidated. In this study, we have assessed E-cadherin protein expression in a well-characterized series of histologically defined ILC (239 cases) with a long-term clinical follow-up to determine the frequency, clinical and biological significance of its expression. E-cadherin-positive ILCs (ILC+) were subsequently examined to assess the expression of component members of the E-cadherin membrane complex (E-cadherin, p120, α, β, and γ-catenins) to determine its integrity.
RESULTS: Thirty-eight ILC cases (16%) showed positive E-cadherin expression (ILC+). Membranous expression of E-cadherin was mainly circumferential with frequent coexisting perimembranous cytoplasmic expression. No association between E-cadherin expression and any of the clinicopathologic variables, immunophenotype, or tumor behavior was identified, apart from an association with lobular histologic subtype and vascular invasion. Analysis of the E-cadherin-catenin complex showed abnormal expression of one or more of the catenin complex members in the majority of cases. The most frequent observation was the diffuse cytoplasmic expression of catenins, in particular p120, which showed similar expression to that reported in E-cadherin-negative ILCs.
CONCLUSIONS: These results provide evidence that E-cadherin is expressed in a proportion of ILC, however, unlike ductal carcinoma, its expression seems to be of limited significance and it is usually associated with evidence of impaired integrity of the E-cadherin-catenin membrane complex. Our data offer a possible explanation for the presence but lack functionality of E-cadherin in some cases of ILC and imply that immunohistochemical expression of E-cadherin per se in ILC histologic phenotypic tumors should not preclude its diagnosis.

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Year:  2010        PMID: 20871222     DOI: 10.1097/PAS.0b013e3181f01916

Source DB:  PubMed          Journal:  Am J Surg Pathol        ISSN: 0147-5185            Impact factor:   6.394


  30 in total

1.  E-cadherin immunohistochemical expression in invasive lobular carcinoma of the breast: correlation with morphology and CDH1 somatic alterations.

Authors:  Anne Grabenstetter; Abhinita S Mohanty; Satshil Rana; Ahmet Zehir; A Rose Brannon; Timothy M D'Alfonso; Deborah F DeLair; Lee K Tan; Dara S Ross
Journal:  Hum Pathol       Date:  2020-06-26       Impact factor: 3.466

Review 2.  Loss of E-Cadherin-Dependent Cell-Cell Adhesion and the Development and Progression of Cancer.

Authors:  Heather C Bruner; Patrick W B Derksen
Journal:  Cold Spring Harb Perspect Biol       Date:  2018-03-01       Impact factor: 10.005

3.  Breast cancer subtypes: morphologic and biologic characterization.

Authors:  Shahla Masood
Journal:  Womens Health (Lond)       Date:  2016-01-12

4.  Loss of E-cadherin Enhances IGF1-IGF1R Pathway Activation and Sensitizes Breast Cancers to Anti-IGF1R/InsR Inhibitors.

Authors:  Alison M Nagle; Kevin M Levine; Nilgun Tasdemir; Julie A Scott; Kara Burlbaugh; Justin Kehm; Tiffany A Katz; David N Boone; Britta M Jacobsen; Jennifer M Atkinson; Steffi Oesterreich; Adrian V Lee
Journal:  Clin Cancer Res       Date:  2018-06-25       Impact factor: 12.531

Review 5.  Molecular Classification of Breast Carcinoma: From Traditional, Old-Fashioned Way to A New Age, and A New Way.

Authors:  Nuket Eliyatkın; Evrim Yalçın; Baha Zengel; Safiye Aktaş; Enver Vardar
Journal:  J Breast Health       Date:  2015-04-01

Review 6.  American Registry of Pathology Expert Opinions: The Spectrum of Lobular Carcinoma in Situ: Diagnostic Features and Clinical Implications.

Authors:  Stuart J Schnitt; Edi Brogi; Yunn-Yi Chen; Tari A King; Sunil R Lakhani
Journal:  Ann Diagn Pathol       Date:  2020-02-15       Impact factor: 2.090

7.  Glucocorticoids and histone deacetylase inhibitors cooperate to block the invasiveness of basal-like breast cancer cells through novel mechanisms.

Authors:  M E Law; P E Corsino; S C Jahn; B J Davis; S Chen; B Patel; K Pham; J Lu; B Sheppard; P Nørgaard; J Hong; P Higgins; J-S Kim; H Luesch; B K Law
Journal:  Oncogene       Date:  2012-04-30       Impact factor: 9.867

8.  Truncated ASPP2 Drives Initiation and Progression of Invasive Lobular Carcinoma via Distinct Mechanisms.

Authors:  Koen Schipper; Anne Paulien Drenth; Eline van der Burg; Samuel Cornelissen; Sjoerd Klarenbeek; Micha Nethe; Jos Jonkers
Journal:  Cancer Res       Date:  2020-02-14       Impact factor: 12.701

Review 9.  p120 catenin: an essential regulator of cadherin stability, adhesion-induced signaling, and cancer progression.

Authors:  Antonis Kourtidis; Siu P Ngok; Panos Z Anastasiadis
Journal:  Prog Mol Biol Transl Sci       Date:  2013       Impact factor: 3.622

10.  E-cadherin expression: a diagnostic utility for differentiating breast carcinomas with ductal and lobular morphologies.

Authors:  Kanthilatha Pai; Poornima Baliga; Bishwo Lal Shrestha
Journal:  J Clin Diagn Res       Date:  2013-03-18
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