Literature DB >> 20837002

Serum soluble CD36, assessed by a novel monoclonal antibody-based sandwich ELISA, predicts cardiovascular mortality in dialysis patients.

Michal Chmielewski1, Ann-Christin Bragfors-Helin, Peter Stenvinkel, Bengt Lindholm, Björn Anderstam.   

Abstract

AIM: Accelerated atherosclerosis is a characteristic feature of chronic kidney disease (CKD). CD36 is a scavenger receptor which contributes to foam cell formation, an early crucial step in atherosclerosis development. Recently, a soluble form of CD36 (sCD36) has been discovered. The aim of the study was to develop an ELISA method for quantitative sCD36 evaluation and to measure it in a cohort of CKD stage 5 patients.
METHOD: A novel monoclonal antibody-based sandwich ELISA for sCD36 evaluation was developed and verified by repeated optimization procedures. Serum concentration of sCD36 was then analyzed in a cohort of 228 CKD stage 5 patients prior to dialysis initiation. Additionally, samples from a control group of 73 healthy, age and gender-matched subjects were evaluated.
RESULTS: The novel CD36 ELISA assay had a recovery of at least 90%, and intra- and inter-assay variability of 6 and 11%, respectively. Concentration of serum sCD36 in CKD patients was significantly increased as compared to controls, and associated with the use of HMG-CoA reductase inhibitors (statins) and the presence of diabetes mellitus (DM). Patients above the 75th percentile of sCD36 concentration were at increased risk of 3-year cardiovascular mortality, as compared to the rest of the cohort [HR 2.85 (1.09-7.59) p=0.03].
CONCLUSION: For the first time, sCD36 was assessed quantitatively in a group of patients and showed associations with DM, CKD, and statin use. Furthermore, the concentration of sCD36 predicted cardiovascular mortality in CKD stage 5 patients.
Copyright © 2010 Elsevier B.V. All rights reserved.

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Year:  2010        PMID: 20837002     DOI: 10.1016/j.cca.2010.09.009

Source DB:  PubMed          Journal:  Clin Chim Acta        ISSN: 0009-8981            Impact factor:   3.786


  15 in total

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Review 3.  CD36 in chronic kidney disease: novel insights and therapeutic opportunities.

Authors:  Xiaochun Yang; Daryl M Okamura; Xifeng Lu; Yaxi Chen; John Moorhead; Zac Varghese; Xiong Z Ruan
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4.  Genetic variants, gene expression, and soluble CD36 analysis in acute coronary syndrome: Differential protein concentration between ST-segment elevation myocardial infarction and unstable angina.

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Authors:  Mohammad J Alkhatatbeh; Nehad M Ayoub; Nizar M Mhaidat; Nesreen A Saadeh; Lisa F Lincz
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8.  Circulating CD36 is increased in hyperlipidemic mice: Cellular sources and triggers of release.

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9.  The origin of circulating CD36 in type 2 diabetes.

Authors:  M J Alkhatatbeh; A K Enjeti; S Acharya; R F Thorne; L F Lincz
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10.  Circulating CD36 is reduced in HNF1A-MODY carriers.

Authors:  Siobhan Bacon; Ma P Kyithar; Jasmin Schmid; Andre Costa Pozza; Aase Handberg; Maria M Byrne
Journal:  PLoS One       Date:  2013-09-12       Impact factor: 3.240

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