Literature DB >> 20825370

The NADH-ubiquinone oxidoreductase 1 alpha subcomplex 5 (NDUFA5) gene variants are associated with autism.

T Marui1, I Funatogawa, S Koishi, K Yamamoto, H Matsumoto, O Hashimoto, S Jinde, H Nishida, T Sugiyama, K Kasai, K Watanabe, Y Kano, N Kato.   

Abstract

OBJECTIVE: Autism appears to have a strong genetic component. The product of the NADH-ubiquinone oxidoreductase 1 alpha subcomplex 5 (NDUFA5) gene is included in the mitochondrial electron transport chain.
METHOD: We performed a case-control study of 235 patients with autism and 214 controls and examined three single-nucleotide polymorphisms (SNPs) within this gene in a Japanese population. We then conducted a transmission disequilibrium test (TDT) analysis in 148 autistic trios.
RESULTS: In the case-control study, two SNPs (rs12666974 and rs3779262) showed a significant association with autism (P=0.00064 and 0.00046 respectively). Furthermore, a haplotype containing these two SNPs showed a significant association (P-global=0.0013, individual haplotype A-A: P=0.010). In TDT analysis, the global and A-A haplotype P-values also indicated significant associations. Minor allele and genotype frequencies were decreased in the autistic subjects.
CONCLUSION: We found significant association between the NDFA5 gene and autism.
© 2010 John Wiley & Sons A/S.

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Year:  2010        PMID: 20825370     DOI: 10.1111/j.1600-0447.2010.01600.x

Source DB:  PubMed          Journal:  Acta Psychiatr Scand        ISSN: 0001-690X            Impact factor:   6.392


  8 in total

1.  Common DNA methylation alterations in multiple brain regions in autism.

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2.  Using extended pedigrees to identify novel autism spectrum disorder (ASD) candidate genes.

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3.  PPARα modulates gene expression profiles of mitochondrial energy metabolism in oral tumorigenesis.

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Review 4.  Evidence of Mitochondrial Dysfunction in Autism: Biochemical Links, Genetic-Based Associations, and Non-Energy-Related Mechanisms.

Authors:  Keren K Griffiths; Richard J Levy
Journal:  Oxid Med Cell Longev       Date:  2017-05-29       Impact factor: 6.543

5.  Hepatocyte miR-33a mediates mitochondrial dysfunction and hepatosteatosis by suppressing NDUFA5.

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6.  Identification of muscle-specific candidate genes in Simmental beef cattle using imputed next generation sequencing.

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7.  Role of NAD(+), Oxidative Stress, and Tryptophan Metabolism in Autism Spectrum Disorders.

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Review 8.  Clinical and Molecular Characteristics of Mitochondrial Dysfunction in Autism Spectrum Disorder.

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  8 in total

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