Literature DB >> 20818955

Increased myogenic responses of resistance-sized mesenteric arteries after reduced uterine perfusion pressure in pregnant rats.

Rolando J J Ramirez1, Julianna Debrah, Jacqueline Novak.   

Abstract

OBJECTIVE: A central component of preeclampsia is a reduction in utero-placental perfusion. We tested the hypothesis that vascular reactivity of second - order mesenteric arteries would be increased in a pregnant rat with reduced uterine perfusion pressure (RUPP).
METHODS: Pregnant 10-12 week old Sprague-Dawley rats underwent RUPP surgery on gestational day 14, and experiments were carried out on day 20. SHAM operated animals were used as controls. Resistance caliber mesenteric arteries (200-250 μm) were isolated, myogenic reactivity and responses to vasoconstrictor and vasodilator agonists were assessed utilizing a pressurized arteriograph system.
RESULTS: RUPP resulted in maternal hypertension and reductions in fetal number and weight. Resistance caliber mesenteric arteries (200-250 μm) were isolated, myogenic reactivity and responses to vasoconstrictor and vasodilator agonists were assessed utilizing a pressurized arteriograph system. Myogenic reactivity responses were normalized as a percent change in vessel diameter from an initial diameter at 20 mmHg. The mesenteric arteries from RUPP animals exhibited a significant increase in myogenic reactivity compared to SHAM controls (p < 0.05). This increased myogenicity was reversed with prostaglandin inhibition, suggesting a role for a vasoconstrictor prostaglandin. In addition to alterations in myogenic reactivity, resistance-sized arteries from RUPP animals have decreased responses to nitric oxide (NO) as evidenced by decreases in responses to methacholine (ME; P < 0.05) and no change in myogenic reactivity after NO synthase blockade.
CONCLUSION: RUPP alters the behavior of resistance caliber arteries to favor a more contractile phenotype with decreased in NO responses, which is similar to what is seen in preeclampsia.

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Year:  2010        PMID: 20818955     DOI: 10.3109/10641950903322923

Source DB:  PubMed          Journal:  Hypertens Pregnancy        ISSN: 1064-1955            Impact factor:   2.108


  5 in total

Review 1.  A model of preeclampsia in rats: the reduced uterine perfusion pressure (RUPP) model.

Authors:  Jing Li; Babbette LaMarca; Jane F Reckelhoff
Journal:  Am J Physiol Heart Circ Physiol       Date:  2012-04-20       Impact factor: 4.733

2.  Increased Myogenic Reactivity of Uterine Arteries from Pregnant Rats with Reduced Uterine Perfusion Pressure.

Authors:  John J Reho; Jonathan D Toot; Jennifer Peck; Jacqueline Novak; Yang H Yun; Rolando J Ramirez
Journal:  Pregnancy Hypertens       Date:  2011-12-13       Impact factor: 2.899

3.  Characterisation of the Selective Reduced Uteroplacental Perfusion (sRUPP) Model of Preeclampsia.

Authors:  J S Morton; J Levasseur; E Ganguly; A Quon; R Kirschenman; J R B Dyck; G M Fraser; S T Davidge
Journal:  Sci Rep       Date:  2019-07-02       Impact factor: 4.379

4.  Sildenafil Therapy Normalizes the Aberrant Metabolomic Profile in the Comt(-/-) Mouse Model of Preeclampsia/Fetal Growth Restriction.

Authors:  Joanna L Stanley; Karolina Sulek; Irene J Andersson; Sandra T Davidge; Louise C Kenny; Colin P Sibley; Rupasri Mandal; David S Wishart; David I Broadhurst; Philip N Baker
Journal:  Sci Rep       Date:  2015-12-15       Impact factor: 4.379

5.  Downregulation of microvascular endothelial type B endothelin receptor is a central vascular mechanism in hypertensive pregnancy.

Authors:  Marc Q Mazzuca; Wei Li; Ossama M Reslan; Peng Yu; Karina M Mata; Raouf A Khalil
Journal:  Hypertension       Date:  2014-06-09       Impact factor: 10.190

  5 in total

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