Literature DB >> 20818652

Subconjunctival carboplatin and systemic topotecan treatment in preclinical models of retinoblastoma.

Katie M Nemeth1, Sara Federico, Angel M Carcaboso, Ying Shen, Paula Schaiquevich, Jiakun Zhang, Merrill Egorin, Clinton Stewart, Michael A Dyer.   

Abstract

BACKGROUND: The authors demonstrated previously that the combination of topotecan (TPT) and carboplatin (CBP) was more effective than current chemotherapeutic combinations used to treat retinoblastoma in an orthotopic xenograft model. However, systemic coadministration of these agents is not ideal, because both agents cause dose-limiting myelosuppression in children.
METHODS: To overcome the toxicity associated with systemic TPT and CBP, the authors explored subconjunctival delivery of TPT or CBP in an orthotopic xenograft model and in a genetic mouse model of retinoblastoma (Chx10-Cre;Rb(lox/lox);p107(-/-);p53(lox/lox)). The effects of combined subconjunctival CBP (CBP(subcon)) and systemic TPT (TPT(syst)) were compared with the effects of combined TPT(subcon) and CBP(syst.) at clinically relevant dosages.
RESULTS: Pharmacokinetic and tumor-response studies, including analyses of ocular and hematopoietic toxicity, revealed that CBP(subcon)/TPT(syst) was more effective and had fewer side effects than TPT(subcon)/CBP(syst).
CONCLUSIONS: For the first time, retinoblastoma was ablated and long-term vision was preserved in a mouse model by using a clinically relevant chemotherapy regimen. These results eventually may be translated into a clinical trial for children with this debilitating cancer.
Copyright © 2010 American Cancer Society.

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Year:  2010        PMID: 20818652      PMCID: PMC3000447          DOI: 10.1002/cncr.25574

Source DB:  PubMed          Journal:  Cancer        ISSN: 0008-543X            Impact factor:   6.860


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4.  Phase I study of combination topotecan and carboplatin in pediatric solid tumors.

Authors:  Uma H Athale; Clinton Stewart; John F Kuttesch; Albert Moghrabi; William Meyer; Charles Pratt; Amar Gajjar; Richard L Heideman
Journal:  J Clin Oncol       Date:  2002-01-01       Impact factor: 44.544

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6.  Inactivation of the p53 pathway in retinoblastoma.

Authors:  Nikia A Laurie; Stacy L Donovan; Chie-Schin Shih; Jiakun Zhang; Nicholas Mills; Christine Fuller; Amina Teunisse; Suzanne Lam; Yolande Ramos; Adithi Mohan; Dianna Johnson; Matthew Wilson; Carlos Rodriguez-Galindo; Micaela Quarto; Sarah Francoz; Susan M Mendrysa; R Kiplin Guy; Jean-Christophe Marine; Aart G Jochemsen; Michael A Dyer
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7.  A phase I/II study of subconjunctival carboplatin for intraocular retinoblastoma.

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Review 10.  Use of preclinical models to improve treatment of retinoblastoma.

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6.  Ocular Salvage and Vision Preservation Using a Topotecan-Based Regimen for Advanced Intraocular Retinoblastoma.

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Review 7.  Progress in Small Molecule Therapeutics for the Treatment of Retinoblastoma.

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8.  Retinoblastoma tumor cell proliferation is negatively associated with an immune gene expression signature and increased immune cells.

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Review 10.  Lessons from Retinoblastoma: Implications for Cancer, Development, Evolution, and Regenerative Medicine.

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