Literature DB >> 20811817

Wnt/beta-catenin is involved in the proliferation of hippocampal neural stem cells induced by hypoxia.

X-P Cui1, Y Xing, J-M Chen, S-W Dong, D-J Ying, D T Yew.   

Abstract

BACKGROUND AND AIM: Beta-catenin, as a major effector molecule in the canonical Wnt signaling pathway, could regulate adult neurogenesis. Here, the role of Wnt/β-catenin signaling pathway in the proliferation of hippocampal neural stem cells (NSCs) induced by hypoxia was investigated.
METHODS: The hippocampal NSCs of neonatal green fluorescent protein transgenic mice on day 0 were cultured in hypoxia (5% O(2)) and traditional O(2) (20% O(2)). The expression of β-catenin, p-GSK-3β, and cyclinD1 in NSCs was measured under hypoxia or traditional O(2) by western blotting. NSCs were electroporated with pTOPFLASH reporter in different conditions and the LEF/TCF-dependent luciferase activity was assayed.
RESULTS: Hypoxia increased the proliferation and reduced the apoptosis of hippocampal NSCs. NSCs proliferation was inhibited by transfecting with pAxin, whereas promoted by transfecting with pβ-catenin.
CONCLUSION: Hypoxia could enhance the proliferation of hippocampal NSCs and β-catenin contributed to this action.

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Year:  2010        PMID: 20811817     DOI: 10.1007/s11845-010-0566-3

Source DB:  PubMed          Journal:  Ir J Med Sci        ISSN: 0021-1265            Impact factor:   1.568


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