Literature DB >> 20798379

Cardiovascular pathology in Hutchinson-Gilford progeria: correlation with the vascular pathology of aging.

Michelle Olive1, Ingrid Harten, Richard Mitchell, Jeanette K Beers, Karima Djabali, Kan Cao, Michael R Erdos, Cecilia Blair, Birgit Funke, Leslie Smoot, Marie Gerhard-Herman, Jason T Machan, Robert Kutys, Renu Virmani, Francis S Collins, Thomas N Wight, Elizabeth G Nabel, Leslie B Gordon.   

Abstract

OBJECTIVE: Children with Hutchinson-Gilford progeria syndrome (HGPS) exhibit dramatically accelerated cardiovascular disease (CVD), causing death from myocardial infarction or stroke between the ages of 7 and 20 years. We undertook the first histological comparative evaluation between genetically confirmed HGPS and the CVD of aging. METHODS AND
RESULTS: We present structural and immunohistological analysis of cardiovascular tissues from 2 children with HGPS who died of myocardial infarction. Both had features classically associated with the atherosclerosis of aging, as well as arteriolosclerosis of small vessels. In addition, vessels exhibited prominent adventitial fibrosis, a previously undescribed feature of HGPS. Importantly, although progerin was detected at higher rates in the HGPS coronary arteries, it was also present in non-HGPS individuals. Between the ages of 1 month and 97 years, progerin staining increased an average of 3.34% per year (P<0.0001) in coronary arteries.
CONCLUSIONS: We find concordance among many aspects of cardiovascular pathology in both HGPS and geriatric patients. HGPS generates a more prominent adventitial fibrosis than typical CVD. Vascular progerin generation in young non-HGPS individuals, which significantly increases throughout life, strongly suggests that progerin has a role in cardiovascular aging of the general population.

Entities:  

Mesh:

Substances:

Year:  2010        PMID: 20798379      PMCID: PMC2965471          DOI: 10.1161/ATVBAHA.110.209460

Source DB:  PubMed          Journal:  Arterioscler Thromb Vasc Biol        ISSN: 1079-5642            Impact factor:   8.311


  32 in total

Review 1.  Farnesylated lamins, progeroid syndromes and farnesyl transferase inhibitors.

Authors:  Antonio E Rusiñol; Michael S Sinensky
Journal:  J Cell Sci       Date:  2006-08-15       Impact factor: 5.285

Review 2.  Ageing of the conduit arteries.

Authors:  S E Greenwald
Journal:  J Pathol       Date:  2007-01       Impact factor: 7.996

Review 3.  Topological determinants and consequences of adventitial responses to arterial wall injury.

Authors:  Jean-Baptiste Michel; Olivier Thaunat; Xavier Houard; Olivier Meilhac; Giuseppina Caligiuri; Antonino Nicoletti
Journal:  Arterioscler Thromb Vasc Biol       Date:  2007-03-29       Impact factor: 8.311

4.  Mutant nuclear lamin A leads to progressive alterations of epigenetic control in premature aging.

Authors:  Dale K Shumaker; Thomas Dechat; Alexander Kohlmaier; Stephen A Adam; Matthew R Bozovsky; Michael R Erdos; Maria Eriksson; Anne E Goldman; Satya Khuon; Francis S Collins; Thomas Jenuwein; Robert D Goldman
Journal:  Proc Natl Acad Sci U S A       Date:  2006-05-31       Impact factor: 11.205

Review 5.  Hutchinson-Gilford progeria syndrome: review of the phenotype.

Authors:  Raoul C M Hennekam
Journal:  Am J Med Genet A       Date:  2006-12-01       Impact factor: 2.802

Review 6.  The immune response in atherosclerosis: a double-edged sword.

Authors:  Göran K Hansson; Peter Libby
Journal:  Nat Rev Immunol       Date:  2006-06-16       Impact factor: 53.106

Review 7.  Cross-link breakers as a new therapeutic approach to cardiovascular disease.

Authors:  D Susic
Journal:  Biochem Soc Trans       Date:  2007-11       Impact factor: 5.407

8.  A lamin A protein isoform overexpressed in Hutchinson-Gilford progeria syndrome interferes with mitosis in progeria and normal cells.

Authors:  Kan Cao; Brian C Capell; Michael R Erdos; Karima Djabali; Francis S Collins
Journal:  Proc Natl Acad Sci U S A       Date:  2007-03-14       Impact factor: 11.205

9.  Phenotype and course of Hutchinson-Gilford progeria syndrome.

Authors:  Melissa A Merideth; Leslie B Gordon; Sarah Clauss; Vandana Sachdev; Ann C M Smith; Monique B Perry; Carmen C Brewer; Christopher Zalewski; H Jeffrey Kim; Beth Solomon; Brian P Brooks; Lynn H Gerber; Maria L Turner; Demetrio L Domingo; Thomas C Hart; Jennifer Graf; James C Reynolds; Andrea Gropman; Jack A Yanovski; Marie Gerhard-Herman; Francis S Collins; Elizabeth G Nabel; Richard O Cannon; William A Gahl; Wendy J Introne
Journal:  N Engl J Med       Date:  2008-02-07       Impact factor: 91.245

10.  The mutant form of lamin A that causes Hutchinson-Gilford progeria is a biomarker of cellular aging in human skin.

Authors:  Dayle McClintock; Desiree Ratner; Meepa Lokuge; David M Owens; Leslie B Gordon; Francis S Collins; Karima Djabali
Journal:  PLoS One       Date:  2007-12-05       Impact factor: 3.240

View more
  155 in total

1.  Replication factor C1, the large subunit of replication factor C, is proteolytically truncated in Hutchinson-Gilford progeria syndrome.

Authors:  Hui Tang; Benjamin Hilton; Phillip R Musich; Ding Zhi Fang; Yue Zou
Journal:  Aging Cell       Date:  2012-01-13       Impact factor: 9.304

Review 2.  Stem cells and the vasculature.

Authors:  Victoria L Bautch
Journal:  Nat Med       Date:  2011-11-07       Impact factor: 53.440

Review 3.  Skin Disease in Laminopathy-Associated Premature Aging.

Authors:  Tomás McKenna; Agustín Sola Carvajal; Maria Eriksson
Journal:  J Invest Dermatol       Date:  2015-07-29       Impact factor: 8.551

Review 4.  The adventitia: a progenitor cell niche for the vessel wall.

Authors:  Mark W Majesky; Xiu Rong Dong; Virginia Hoglund; Gunter Daum; William M Mahoney
Journal:  Cells Tissues Organs       Date:  2011-10-14       Impact factor: 2.481

5.  Coronary artery disease in a Werner syndrome-like form of progeria characterized by low levels of progerin, a splice variant of lamin A.

Authors:  Fuki M Hisama; Davor Lessel; Dru Leistritz; Katrin Friedrich; Kim L McBride; Matthew T Pastore; Gary S Gottesman; Bidisha Saha; George M Martin; Christian Kubisch; Junko Oshima
Journal:  Am J Med Genet A       Date:  2011-11-07       Impact factor: 2.802

Review 6.  Nuclear mechanics in disease.

Authors:  Monika Zwerger; Chin Yee Ho; Jan Lammerding
Journal:  Annu Rev Biomed Eng       Date:  2011-08-15       Impact factor: 9.590

7.  Truncated prelamin A expression in HGPS-like patients: a transcriptional study.

Authors:  Florian Barthélémy; Claire Navarro; Racha Fayek; Nathalie Da Silva; Patrice Roll; Sabine Sigaudy; Junko Oshima; Gisèle Bonne; Kyriaki Papadopoulou-Legbelou; Athanasios E Evangeliou; Martha Spilioti; Martine Lemerrer; Ron A Wevers; Eva Morava; Andrée Robaglia-Schlupp; Nicolas Lévy; Marc Bartoli; Annachiara De Sandre-Giovannoli
Journal:  Eur J Hum Genet       Date:  2015-02-04       Impact factor: 4.246

8.  Progerin sequestration of PCNA promotes replication fork collapse and mislocalization of XPA in laminopathy-related progeroid syndromes.

Authors:  Benjamin A Hilton; Ji Liu; Brian M Cartwright; Yiyong Liu; Maya Breitman; Youjie Wang; Rowdy Jones; Hui Tang; Antonio Rusinol; Phillip R Musich; Yue Zou
Journal:  FASEB J       Date:  2017-05-17       Impact factor: 5.191

Review 9.  Endothelial PPARγ Is Crucial for Averting Age-Related Vascular Dysfunction by Stalling Oxidative Stress and ROCK.

Authors:  Md Sahab Uddin; Md Tanvir Kabir; Md Jakaria; Abdullah Al Mamun; Kamal Niaz; Md Shah Amran; George E Barreto; Ghulam Md Ashraf
Journal:  Neurotox Res       Date:  2019-05-04       Impact factor: 3.911

Review 10.  Mechanisms of Vascular Aging.

Authors:  Zoltan Ungvari; Stefano Tarantini; Anthony J Donato; Veronica Galvan; Anna Csiszar
Journal:  Circ Res       Date:  2018-09-14       Impact factor: 17.367

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.