Literature DB >> 20718100

Pain: new familial pain syndrome caused by TRPA1 mutation.

Heather Wood.   

Abstract

Entities:  

Year:  2010        PMID: 20718100     DOI: 10.1038/nrneurol.2010.97

Source DB:  PubMed          Journal:  Nat Rev Neurol        ISSN: 1759-4758            Impact factor:   42.937


× No keyword cloud information.
  1 in total

1.  A gain-of-function mutation in TRPA1 causes familial episodic pain syndrome.

Authors:  Barbara Kremeyer; Francisco Lopera; James J Cox; Aliakmal Momin; Francois Rugiero; Steve Marsh; C Geoffrey Woods; Nicholas G Jones; Kathryn J Paterson; Florence R Fricker; Andrés Villegas; Natalia Acosta; Nicolás G Pineda-Trujillo; Juan Diego Ramírez; Julián Zea; Mari-Wyn Burley; Gabriel Bedoya; David L H Bennett; John N Wood; Andrés Ruiz-Linares
Journal:  Neuron       Date:  2010-06-10       Impact factor: 17.173

  1 in total
  3 in total

Review 1.  The transient receptor potential channel TRPA1: from gene to pathophysiology.

Authors:  Bernd Nilius; Giovanni Appendino; Grzegorz Owsianik
Journal:  Pflugers Arch       Date:  2012-09-22       Impact factor: 3.657

Review 2.  TRP channels: a journey towards a molecular understanding of pain.

Authors:  Tamara Rosenbaum; Sara L Morales-Lázaro; León D Islas
Journal:  Nat Rev Neurosci       Date:  2022-07-13       Impact factor: 38.755

3.  Effects of N-Glycosylation of the human cation channel TRPA1 on agonist-sensitivity.

Authors:  Timothy James Egan; Mario A Acuña; Marcy Zenobi-Wong; Hanns Ulrich Zeilhofer; David Urech
Journal:  Biosci Rep       Date:  2016-08-31       Impact factor: 3.840

  3 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.