Literature DB >> 20688215

Human peripheral blood-derived CD31+ cells have robust angiogenic and vasculogenic properties and are effective for treating ischemic vascular disease.

Sung-Whan Kim1, Hyongbum Kim, Hyun-Jai Cho, Jung-Uek Lee, Rebecca Levit, Young-sup Yoon.   

Abstract

OBJECTIVES: This study aimed to determine if CD31 is a novel marker of a circulating angio-vasculogenic cell population and to establish the cells' therapeutic effects on experimental ischemia.
BACKGROUND: Emerging evidence suggested that therapeutic mechanisms underlying various bone marrow-derived cells are due to paracrine effects. Furthermore, the vasculogenic potential of these cells is under debate. CD31 is a well-known marker for endothelial cells but is also expressed in a fraction of peripheral blood (PB) mononuclear cells.
METHODS: CD31(+) cells were isolated from human PB by magnetic-activated cell sorting. The gene expression profile was examined by deoxyribonucleic acid microarray and real-time reverse transcriptase polymerase chain reaction. Various in vitro endothelial differentiation or vasculogenic assays were conducted. Finally, cells were directly implanted into a mouse hind limb ischemia model to test angiogenic-vasculogenic and therapeutic effects.
RESULTS: Fluorescent-activated cell sorter analysis revealed that PB-CD31(+) cells exhibited endothelial and hematopoietic stem/progenitor markers. CD31(+) cells had higher levels of expression of proangiogenic genes on microarray and real-time reverse transcriptase polymerase chain reaction and generated higher numbers of endothelial progenitor cells than CD31(-) cells did. CD31(+) cells spontaneously formed vascular tubelike structures and exhibited an endothelial cell phenotype in vitro. In a hind limb ischemia model, CD31(+) cell transplantation augmented blood perfusion and prevented limb loss. Both angiogenic cytokines and capillary density were increased, suggesting CD31(+) cells augmented neovascularization.
CONCLUSIONS: CD31 is a novel marker that designates circulating angiogenic and vasculogenic cells. These cells are easily isolated from human PB and thus are a novel candidate for treatment of ischemic cardiovascular disease. Copyright (c) 2010 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved.

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Year:  2010        PMID: 20688215      PMCID: PMC2917842          DOI: 10.1016/j.jacc.2010.01.070

Source DB:  PubMed          Journal:  J Am Coll Cardiol        ISSN: 0735-1097            Impact factor:   24.094


  30 in total

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Review 3.  Regulation of endothelial cell survival and apoptosis during angiogenesis.

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9.  Intramyocardial transplantation of autologous endothelial progenitor cells for therapeutic neovascularization of myocardial ischemia.

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10.  Role of host tissues for sustained humoral effects after endothelial progenitor cell transplantation into the ischemic heart.

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1.  Hypoxically preconditioned human peripheral blood mononuclear cells improve blood flow in hindlimb ischemia xenograft model.

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Review 3.  Revisiting cardiovascular regeneration with bone marrow-derived angiogenic and vasculogenic cells.

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4.  Bone Marrow-Derived Mesenchymal Stem Cells Improve Diabetic Neuropathy by Direct Modulation of Both Angiogenesis and Myelination in Peripheral Nerves.

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6.  Receptors for non-MHC ligands contribute to uterine natural killer cell activation during pregnancy in mice.

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7.  High proliferative potential endothelial colony-forming cells contribute to hypoxia-induced pulmonary artery vasa vasorum neovascularization.

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Review 8.  Critical reevaluation of endothelial progenitor cell phenotypes for therapeutic and diagnostic use.

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9.  Induction of pluripotency in bone marrow mononuclear cells via polyketal nanoparticle-mediated delivery of mature microRNAs.

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10.  Circulating CD31+ leukocyte frequency is associated with cardiovascular risk factors.

Authors:  Yin Ge; Susan Cheng; Martin G Larson; Anahita Ghorbani; Roderick P Martin; Rachael J Klein; Christopher J O'Donnell; Ramachandran S Vasan; Stanley Y Shaw; Thomas J Wang; Kenneth S Cohen
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