Literature DB >> 20682976

Epithelial-mesenchymal transition in EGFR-TKI acquired resistant lung adenocarcinoma.

Hidetaka Uramoto1, Teruo Iwata, Takamitsu Onitsuka, Hidehiko Shimokawa, Takeshi Hanagiri, Tsunehiro Oyama.   

Abstract

BACKGROUND: The epithelial to mesenchymal transition (EMT) may well play a part in determining the sensitivity to epidermal growth factor receptor (EGFR) tyrosine kinase inhibitor (TKI). However to date, no study has investigated the association between the EMT status and acquired resistance using cancer specimens. PATIENTS AND METHODS: Immunohistochemical (IHC) staining was used to analyse the protein expression of epithelial and mesenchymal markers in tumour samples from lung adenocarcinoma patients. Mutations in the EGFR and K-ras gene were also examined.
RESULTS: All patients showed a positive expression of epithelial markers in sensitive tumours. Tumour in 4 (44.4%) out of 9 patients showed down-regulation of epithelial markers or up-regulation of mesenchymal markers. The change in the EMT status between pre-and post-treatment was shown in 2 cases each with and without the T790M mutation.
CONCLUSION: EMT plays a role in approximately half of the cases of resistance to EGFR-TKI, independent of T790M mutation.

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Year:  2010        PMID: 20682976

Source DB:  PubMed          Journal:  Anticancer Res        ISSN: 0250-7005            Impact factor:   2.480


  68 in total

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10.  FLNA promotes chemoresistance of colorectal cancer through inducing epithelial-mesenchymal transition and smad2 signaling pathway.

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