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Literature DB >> 20679071

Editorial: PD-1, a new target for sepsis treatment: better late than never.

Sanna M Goyert, Jack Silver.

Abstract

Entities: Disease Gene

Mesh：

Substances：

Year: 2010 PMID： 20679071 DOI： 10.1189/jlb.0410240

Source DB: PubMed Journal: J Leukoc Biol ISSN： 0741-5400 Impact factor: 4.962

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8 in total

Review 1. A historical perspective on sepsis.

Authors: Peter A Ward; Markus Bosmann
Journal: Am J Pathol Date: 2012-05-26 Impact factor: 4.307

2. Sepsis erodes CD8⁺ memory T cell-protective immunity against an EBV homolog in a 2B4-dependent manner.

Authors: Jianfeng Xie; Rebecca L Crepeau; Ching-Wen Chen; Wenxiao Zhang; Shunsuke Otani; Craig M Coopersmith; Mandy L Ford
Journal: J Leukoc Biol Date: 2019-01-09 Impact factor: 4.962

3. Increased attrition of memory T cells during sepsis requires 2B4.

Authors: Jianfeng Xie; Ching-Wen Chen; Yini Sun; Sonia J Laurie; Wenxiao Zhang; Shunsuke Otani; Gregory S Martin; Craig M Coopersmith; Mandy L Ford
Journal: JCI Insight Date: 2019-05-02

4. Kupffer cells potentiate liver sinusoidal endothelial cell injury in sepsis by ligating programmed cell death ligand-1.

Authors: Noelle A Hutchins; Fei Wang; Yvonne Wang; Chun-Shiang Chung; Alfred Ayala
Journal: J Leukoc Biol Date: 2013-06-13 Impact factor: 4.962

5. Programmed death-1 levels correlate with increased mortality, nosocomial infection and immune dysfunctions in septic shock patients.

Authors: Caroline Guignant; Alain Lepape; Xin Huang; Hakim Kherouf; Laure Denis; Françoise Poitevin; Christophe Malcus; Aurélie Chéron; Bernard Allaouchiche; François Gueyffier; Alfred Ayala; Guillaume Monneret; Fabienne Venet
Journal: Crit Care Date: 2011-03-21 Impact factor: 9.097

Editorial: PD-1, a new target for sepsis treatment: better late than never.

Review 1. A historical perspective on sepsis.

2. Sepsis erodes CD8⁺ memory T cell-protective immunity against an EBV homolog in a 2B4-dependent manner.

3. Increased attrition of memory T cells during sepsis requires 2B4.

4. Kupffer cells potentiate liver sinusoidal endothelial cell injury in sepsis by ligating programmed cell death ligand-1.

5. Programmed death-1 levels correlate with increased mortality, nosocomial infection and immune dysfunctions in septic shock patients.

6. To be, or not to be immunocompetent.

7. Pre-existing malignancy results in increased prevalence of distinct populations of CD4+ T cells during sepsis.

Review 8. Immune checkpoint receptors: homeostatic regulators of immunity.

Editorial: PD-1, a new target for sepsis treatment: better late than never.

Review 1. A historical perspective on sepsis.

2. Sepsis erodes CD8+ memory T cell-protective immunity against an EBV homolog in a 2B4-dependent manner.

3. Increased attrition of memory T cells during sepsis requires 2B4.

4. Kupffer cells potentiate liver sinusoidal endothelial cell injury in sepsis by ligating programmed cell death ligand-1.

5. Programmed death-1 levels correlate with increased mortality, nosocomial infection and immune dysfunctions in septic shock patients.

6. To be, or not to be immunocompetent.

7. Pre-existing malignancy results in increased prevalence of distinct populations of CD4+ T cells during sepsis.

Review 8. Immune checkpoint receptors: homeostatic regulators of immunity.

2. Sepsis erodes CD8⁺ memory T cell-protective immunity against an EBV homolog in a 2B4-dependent manner.