Literature DB >> 20675165

Amyloid Aß , cut from APP by ß-secretase BACE1 and γ-secretase, induces apoptosis via opening type-1 porin/VDAC in cell membranes of hypometabolic cells-A basic model for the induction of apoptosis!?

Friedrich P Thinnes.   

Abstract

Entities:  

Mesh：

Substances：

Year:  2010        PMID： 20675165     DOI： 10.1016/j.ymgme.2010.07.007

Source DB:  PubMed          Journal:  Mol Genet Metab        ISSN： 1096-7192            Impact factor:   4.797


× No keyword cloud information.
  5 in total

1.  On GxxxG in N-terminal stretches of type-1 VDAC/porin: critical in vertebrate apoptosis, missing in plants.

Authors:  Friedrich P Thinnes
Journal:  Plant Mol Biol       Date:  2012-03-27       Impact factor: 4.076

2.  Apoptogenic interactions of plasmalemmal type-1 VDAC and Aβ peptides via GxxxG motifs induce Alzheimer's disease - a basic model of apoptosis?

Authors:  Friedrich P Thinnes
Journal:  Wien Med Wochenschr       Date:  2011-04-04

3.  Signalosomes in the brain: relevance in the development of certain neuropathologies such as Alzheimer's disease.

Authors:  Raquel Marin
Journal:  Front Physiol       Date:  2011-06-03       Impact factor: 4.566

4.  Plasmalemmal VDAC-1 corroborated as amyloid Aß-receptor.

Authors:  Friedrich P Thinnes
Journal:  Front Aging Neurosci       Date:  2015-09-30       Impact factor: 5.750

Review 5.  VDAC1 as Pharmacological Target in Cancer and Neurodegeneration: Focus on Its Role in Apoptosis.

Authors:  Andrea Magrì; Simona Reina; Vito De Pinto
Journal:  Front Chem       Date:  2018-04-06       Impact factor: 5.221
  5 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.