Literature DB >> 20668833

Further refinement of the critical minimal genetic region for the imprinting disorder 6q24 transient neonatal diabetes.

L E Docherty1, R L Poole, C J Mattocks, A Lehmann, I K Temple, D J G Mackay.   

Abstract

AIMS/HYPOTHESIS: Transient neonatal diabetes (TND) is associated with overexpression of genes within a critical region on 6q24. This study aims to refine the boundaries of this region to reduce the number of potential candidate genes for 6q24 TND.
METHODS: Fifteen patients with transient neonatal diabetes and submicroscopic chromosome 6 duplications were investigated. The duplications were confirmed by microsatellite analysis and subsequently mapped using tiled chromosome 6 array Comparative Genomic Hybridisation (aCGH) and MLPA. Duplication boundaries were compared to identify the minimal shared region of duplication. These data were then used with available clinical data to identify associations between size of 6q24 duplication and severity of TND phenotype.
RESULTS: Alignment of the minimal region of duplication to the human genome reduced the minimal TND critical region, formerly estimated at 440 kb, to 160-173 kb, revealing PLAGL1 (pleiomorphic adenoma gene-like 1) and HYMAI (imprinted in hydatidiform mole) to be the only genes wholly included therein. Additionally, the complete paternal duplication of a region containing the theoretical protein FAM164B was associated with the severe growth restriction observed in 6q24 duplication patients. CONCLUSIONS/
INTERPRETATION: This study has significantly reduced the critical region associated with 6q24 TND. It has eliminated several previous TND candidate genes, leaving the overlapping imprinted genes PLAGL1 and HYMAI as the only remaining complete candidate genes for 6q24 TND. Moreover, these data provide the first evidence that an additional region, encompassing the theoretical protein FAM164B, may have a critical role in the growth restriction phenotype observed in many 6q24 TND patients.

Entities:  

Mesh:

Year:  2010        PMID: 20668833     DOI: 10.1007/s00125-010-1853-2

Source DB:  PubMed          Journal:  Diabetologia        ISSN: 0012-186X            Impact factor:   10.122


  10 in total

1.  A novel imprinted gene, HYMAI, is located within an imprinted domain on human chromosome 6 containing ZAC.

Authors:  T Arima; R A Drewell; M Oshimura; N Wake; M A Surani
Journal:  Genomics       Date:  2000-08-01       Impact factor: 5.736

2.  The cell cycle control gene ZAC/PLAGL1 is imprinted--a strong candidate gene for transient neonatal diabetes.

Authors:  M Kamiya; H Judson; Y Okazaki; M Kusakabe; M Muramatsu; S Takada; N Takagi; T Arima; N Wake; K Kamimura; K Satomura; R Hermann; D T Bonthron; Y Hayashizaki
Journal:  Hum Mol Genet       Date:  2000-02-12       Impact factor: 6.150

3.  Paternal uniparental disomy of chromosome 6 and transient neonatal diabetes mellitus.

Authors:  R J Gardner; D O Robinson; L Lamont; J P Shield; I K Temple
Journal:  Clin Genet       Date:  1998-12       Impact factor: 4.438

4.  Characterization of the methylation-sensitive promoter of the imprinted ZAC gene supports its role in transient neonatal diabetes mellitus.

Authors:  A Varrault; B Bilanges; D J Mackay; E Basyuk; B Ahr; C Fernandez; D O Robinson; J Bockaert; L Journot
Journal:  J Biol Chem       Date:  2001-04-10       Impact factor: 5.157

5.  Bisulphite sequencing of the transient neonatal diabetes mellitus DMR facilitates a novel diagnostic test but reveals no methylation anomalies in patients of unknown aetiology.

Authors:  Deborah J G Mackay; I Karen Temple; Julian P H Shield; David O Robinson
Journal:  Hum Genet       Date:  2005-01-06       Impact factor: 4.132

6.  An imprinted locus associated with transient neonatal diabetes mellitus.

Authors:  R J Gardner; D J Mackay; A J Mungall; C Polychronakos; R Siebert; J P Shield; I K Temple; D O Robinson
Journal:  Hum Mol Genet       Date:  2000-03-01       Impact factor: 6.150

7.  Relaxation of imprinted expression of ZAC and HYMAI in a patient with transient neonatal diabetes mellitus.

Authors:  D J G Mackay; A-M Coupe; J P H Shield; J N P Storr; I K Temple; D O Robinson
Journal:  Hum Genet       Date:  2002-01-24       Impact factor: 4.132

8.  Impaired glucose homeostasis in transgenic mice expressing the human transient neonatal diabetes mellitus locus, TNDM.

Authors:  Dan Ma; Julian P H Shield; Wendy Dean; Isabelle Leclerc; Claude Knauf; R éMy Burcelin R; Guy A Rutter; Gavin Kelsey
Journal:  J Clin Invest       Date:  2004-08       Impact factor: 14.808

9.  Mutations in ATP-sensitive K+ channel genes cause transient neonatal diabetes and permanent diabetes in childhood or adulthood.

Authors:  Sarah E Flanagan; Ann-Marie Patch; Deborah J G Mackay; Emma L Edghill; Anna L Gloyn; David Robinson; Julian P H Shield; Karen Temple; Sian Ellard; Andrew T Hattersley
Journal:  Diabetes       Date:  2007-04-19       Impact factor: 9.461

10.  Tissue-specific imprinting of the ZAC/PLAGL1 tumour suppressor gene results from variable utilization of monoallelic and biallelic promoters.

Authors:  Elizabeth M Valleley; Sarah F Cordery; David T Bonthron
Journal:  Hum Mol Genet       Date:  2007-03-06       Impact factor: 6.150

  10 in total
  6 in total

1.  Clinical features of three girls with mosaic genome-wide paternal uniparental isodisomy.

Authors:  Jennifer M Kalish; Laura K Conlin; Tricia R Bhatti; Holly A Dubbs; Mary Catherine Harris; Kosuke Izumi; Sogol Mostoufi-Moab; Surabhi Mulchandani; Sulagna Saitta; Lisa J States; Daniel T Swarr; Alisha B Wilkens; Elaine H Zackai; Kristin Zelley; Marisa S Bartolomei; Kim E Nichols; Andrew A Palladino; Nancy B Spinner; Matthew A Deardorff
Journal:  Am J Med Genet A       Date:  2013-06-26       Impact factor: 2.802

Review 2.  Translational implications of the β-cell epigenome in diabetes mellitus.

Authors:  Justin S Johnson; Carmella Evans-Molina
Journal:  Transl Res       Date:  2014-03-12       Impact factor: 7.012

Review 3.  Role of ZAC1 in transient neonatal diabetes mellitus and glucose metabolism.

Authors:  Anke Hoffmann; Dietmar Spengler
Journal:  World J Biol Chem       Date:  2015-08-26

4.  Imprinting at the PLAGL1 domain is contained within a 70-kb CTCF/cohesin-mediated non-allelic chromatin loop.

Authors:  Isabel Iglesias-Platas; Franck Court; Cristina Camprubi; Angela Sparago; Amy Guillaumet-Adkins; Alex Martin-Trujillo; Andrea Riccio; Gudrun E Moore; David Monk
Journal:  Nucleic Acids Res       Date:  2013-01-07       Impact factor: 16.971

5.  Insulin pump therapy in transient neonatal diabetes mellitus.

Authors:  Joong Heum Park; Jeong Hee Kang; Kyu-Hyung Lee; Nam-Hyo Kim; Han-Wook Yoo; Dae-Yeol Lee; Eun-Gyong Yoo
Journal:  Ann Pediatr Endocrinol Metab       Date:  2013-09-30

6.  Syndromic Disorders Caused by Disturbed Human Imprinting

Authors:  Diana Carli; Evelise Riberi; Giovanni Battista Ferrero; Alessandro Mussa
Journal:  J Clin Res Pediatr Endocrinol       Date:  2019-04-10
  6 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.