Literature DB >> 20640406

Inhibition of phosphodiesterase-4 reverses memory deficits produced by Aβ25-35 or Aβ1-40 peptide in rats.

Yu-Fang Cheng1, Chuang Wang, Huan-Bing Lin, Yun-Feng Li, Ying Huang, Jiang-Ping Xu, Han-Ting Zhang.   

Abstract

RATIONALE: Cyclic AMP signaling plays an important role in memory loss associated with Alzheimer's disease (AD). However, little is known about whether inhibition of phosphodiesterase-4 (PDE4), which increases intracellular cAMP, reverses β-amyloid peptide (Aβ)-induced memory deficits.
OBJECTIVE: Experiments were performed to demonstrate the effect of the PDE4 inhibitor rolipram on memory impairment produced by Aβ1-40 (Aβ40) or its core fragment Aβ25-35.
METHODS: We tested memory using Morris water-maze and passive avoidance tasks and examined expression of phosphorylated cAMP response-element binding protein (pCREB) in the hippocampus in rats treated with Aβ25-35 or Aβ40 into bilateral CA1 subregions, with or without rolipram administration.
RESULTS: Aβ25-35 (10 μg/side) increased escape latency during acquisition training and decreased swimming time and distance in the target quadrant in the water-maze probe trial; it also decreased 24-h retention in the passive avoidance paradigm. All these were reversed by chronic administration of rolipram (0.5 mg/kg). Similarly, Aβ40 (4 μg/side) produced memory impairment, as demonstrated by decreased retention in passive avoidance; this was also reversed by repeated treatment with rolipram. In addition, rolipram blocked extinction of memory during the 32-day testing period in the passive avoidance test. Further, Aβ40 decreased pCREB expression in the hippocampus, which was also reversed by rolipram; the changes in pCREB were highly correlated with those in memory.
CONCLUSIONS: These results suggest that the PDE4 inhibitor rolipram reverses cognitive deficits associated with AD most likely via increased cAMP/CREB signaling in the hippocampus; PDE4 could be a target for drugs that improve cognition in AD.

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Year:  2010        PMID: 20640406     DOI: 10.1007/s00213-010-1943-3

Source DB:  PubMed          Journal:  Psychopharmacology (Berl)        ISSN: 0033-3158            Impact factor:   4.530


  81 in total

1.  Time-dependent reorganization of brain circuitry underlying long-term memory storage.

Authors:  B Bontempi; C Laurent-Demir; C Destrade; R Jaffard
Journal:  Nature       Date:  1999-08-12       Impact factor: 49.962

2.  In vitro aggregation facilities beta-amyloid peptide-(25-35)-induced amnesia in the rat.

Authors:  S Delobette; A Privat; T Maurice
Journal:  Eur J Pharmacol       Date:  1997-01-14       Impact factor: 4.432

3.  Action of rolipram on specific PDE4 cAMP phosphodiesterase isoforms and on the phosphorylation of cAMP-response-element-binding protein (CREB) and p38 mitogen-activated protein (MAP) kinase in U937 monocytic cells.

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5.  Induction of a dominant negative CREB transgene specifically blocks long-term memory in Drosophila.

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7.  Development of a femtomolar-acting humanin derivative named colivelin by attaching activity-dependent neurotrophic factor to its N terminus: characterization of colivelin-mediated neuroprotection against Alzheimer's disease-relevant insults in vitro and in vivo.

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Review 2.  PDE4 as a target for cognition enhancement.

Authors:  Wito Richter; Frank S Menniti; Han-Ting Zhang; Marco Conti
Journal:  Expert Opin Ther Targets       Date:  2013-07-25       Impact factor: 6.902

3.  GEBR-7b, a novel PDE4D selective inhibitor that improves memory in rodents at non-emetic doses.

Authors:  O Bruno; E Fedele; J Prickaerts; L A Parker; E Canepa; C Brullo; A Cavallero; E Gardella; A Balbi; C Domenicotti; E Bollen; H J M Gijselaers; T Vanmierlo; K Erb; C L Limebeer; F Argellati; U M Marinari; M A Pronzato; R Ricciarelli
Journal:  Br J Pharmacol       Date:  2011-12       Impact factor: 8.739

Review 4.  Exercise as a Positive Modulator of Brain Function.

Authors:  Karim A Alkadhi
Journal:  Mol Neurobiol       Date:  2017-05-02       Impact factor: 5.590

5.  Inhibition of phosphodiesterase-4 reverses the cognitive dysfunction and oxidative stress induced by Aβ25-35 in rats.

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Journal:  Metab Brain Dis       Date:  2016-02-27       Impact factor: 3.584

6.  Amyloid-β Peptide Is Needed for cGMP-Induced Long-Term Potentiation and Memory.

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Journal:  J Neurosci       Date:  2017-06-16       Impact factor: 6.167

Review 7.  Cyclic nucleotide signaling changes associated with normal aging and age-related diseases of the brain.

Authors:  Michy P Kelly
Journal:  Cell Signal       Date:  2017-11-23       Impact factor: 4.315

8.  β2-adrenergic Agonists Rescue Lysosome Acidification and Function in PSEN1 Deficiency by Reversing Defective ER-to-lysosome Delivery of ClC-7.

Authors:  Ju-Hyun Lee; Devin M Wolfe; Sandipkumar Darji; Mary Kate McBrayer; Daniel J Colacurcio; Asok Kumar; Philip Stavrides; Panaiyur S Mohan; Ralph A Nixon
Journal:  J Mol Biol       Date:  2020-02-24       Impact factor: 5.469

Review 9.  CREB signals as PBMC-based biomarkers of cognitive dysfunction: A novel perspective of the brain-immune axis.

Authors:  Nancy Bartolotti; Orly Lazarov
Journal:  Brain Behav Immun       Date:  2019-01-12       Impact factor: 7.217

Review 10.  Behavioral assays with mouse models of Alzheimer's disease: practical considerations and guidelines.

Authors:  Daniela Puzzo; Linda Lee; Agostino Palmeri; Giorgio Calabrese; Ottavio Arancio
Journal:  Biochem Pharmacol       Date:  2014-01-21       Impact factor: 5.858

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