Literature DB >> 20637369

Immunohistochemical analysis of prostate apoptosis response-4 (Par-4) in Mexican women with breast cancer: a preliminary study.

Luis Fernando Méndez-López1, Pablo Zapata-Benavides, Angel Zavala-Pompa, Miguel Elías Aguado-Barrera, Javier Pacheco-Calleros, Cristina Rodríguez-Padilla, Ricardo Martín Cerda-Flores, Elva Irene Cortés-Gutiérrez, Martha Imelda Dávila-Rodríguez.   

Abstract

BACKGROUND AND AIMS: We undertook this study to compare the expression level of prostate apoptosis response-4 (Par-4) among patient outcome in two groups of women with breast cancer (short and long survival) and two groups without breast cancer (benign lesion and control).
METHODS: We included breast specimens with nonhistological abnormalities (eight samples) as a control group. Semiquantitative and quantitative analysis of immunohistochemical staining by image analysis software were used to study the intensity of Par-4 expression. Both methods produced similar results (p>0.05).
RESULTS: No significant expression of Par-4 was observed in normal breast tissue. Benign lesions and breast cancer tissue showed strong nuclear expression of Par-4, predominantly on epithelial cells and specifically in ductal cells. Par-4 expression was lower in myoepithelial cells and there was no appreciable stromal staining. Significantly less Par-4 reactivity was detected in tissue from patients with a short survival compared with patients with benign lesions and those with a long survival.
CONCLUSIONS: Our findings suggest that a lower expression level of Par-4 is related to an unfavorable prognosis. A larger prospective study of samples of all patient groups with a longer follow-up is needed to validate this finding. Copyright 2010 IMSS. Published by Elsevier Inc. All rights reserved.

Entities:  

Mesh:

Substances:

Year:  2010        PMID: 20637369     DOI: 10.1016/j.arcmed.2010.05.005

Source DB:  PubMed          Journal:  Arch Med Res        ISSN: 0188-4409            Impact factor:   2.235


  5 in total

1.  Revisiting Epithelial Carcinogenesis.

Authors:  Luis Fernando Méndez-López
Journal:  Int J Mol Sci       Date:  2022-07-04       Impact factor: 6.208

2.  Par-4 downregulation promotes breast cancer recurrence by preventing multinucleation following targeted therapy.

Authors:  James V Alvarez; Tien-Chi Pan; Jason Ruth; Yi Feng; Alice Zhou; Dhruv Pant; Joshua S Grimley; Thomas J Wandless; Angela Demichele; Lewis A Chodosh
Journal:  Cancer Cell       Date:  2013-06-13       Impact factor: 31.743

3.  Prostate apoptosis response 4 (PAR4) expression modulates WNT signaling pathways in MCF7 breast cancer cells: A possible mechanism underlying PAR4-mediated docetaxel chemosensitivity.

Authors:  Simone Aparecida de Bessa Garcia; Ana Carolina Pavanelli; Natália Cruz E Melo; Maria Aparecida Nagai
Journal:  Int J Mol Med       Date:  2017-02-21       Impact factor: 4.101

4.  PAR-4 overcomes chemo-resistance in breast cancer cells by antagonizing cIAP1.

Authors:  Haihong Guo; Fabian Treude; Oliver H Krämer; Bernhard Lüscher; Jörg Hartkamp
Journal:  Sci Rep       Date:  2019-06-19       Impact factor: 4.379

5.  Par-4 prevents breast cancer recurrence.

Authors:  Nikhil Hebbar; Tripti Shrestha-Bhattarai; Vivek M Rangnekar
Journal:  Breast Cancer Res       Date:  2013       Impact factor: 6.466

  5 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.