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70 in total

1. PCSK9 reduces the protein levels of the LDL receptor in mouse brain during development and after ischemic stroke.

Authors: Estelle Rousselet; Jadwiga Marcinkiewicz; Jasna Kriz; Ann Zhou; Mary E Hatten; Annik Prat; Nabil G Seidah
Journal: J Lipid Res Date: 2011-04-25 Impact factor: 5.922

2. Rate and equilibrium constants for binding of apo-E HDLc (a cholesterol-induced lipoprotein) and low density lipoproteins to human fibroblasts: evidence for multiple receptor binding of apo-E HDLc.

Authors: R E Pitas; T L Innerarity; K S Arnold; R W Mahley
Journal: Proc Natl Acad Sci U S A Date: 1979-05 Impact factor: 11.205

3. Disruption of LDL but not VLDL clearance in autosomal recessive hypercholesterolemia.

Authors: Christopher Jones; Rita Garuti; Peter Michaely; Wei-Ping Li; Nobuyo Maeda; Jonathan C Cohen; Joachim Herz; Helen H Hobbs
Journal: J Clin Invest Date: 2007-01 Impact factor: 14.808

4. Reversal of defective lysosomal transport in NPC disease ameliorates liver dysfunction and neurodegeneration in the npc1-/- mouse.

Authors: Benny Liu; Stephen D Turley; Dennis K Burns; Anna M Miller; Joyce J Repa; John M Dietschy
Journal: Proc Natl Acad Sci U S A Date: 2009-01-26 Impact factor: 11.205

5. apoE3[K146N/R147W] acts as a dominant negative apoE form that prevents remnant clearance and inhibits the biogenesis of HDL.

Authors: Panagiotis Fotakis; Alexander Vezeridis; Ioannis Dafnis; Angeliki Chroni; Dimitris Kardassis; Vassilis I Zannis
Journal: J Lipid Res Date: 2014-04-28 Impact factor: 5.922

Enhanced binding by cultured human fibroblasts of apo-E-containing lipoproteins as compared with low density lipoproteins.

1. PCSK9 reduces the protein levels of the LDL receptor in mouse brain during development and after ischemic stroke.

2. Rate and equilibrium constants for binding of apo-E HDLc (a cholesterol-induced lipoprotein) and low density lipoproteins to human fibroblasts: evidence for multiple receptor binding of apo-E HDLc.

3. Disruption of LDL but not VLDL clearance in autosomal recessive hypercholesterolemia.

4. Reversal of defective lysosomal transport in NPC disease ameliorates liver dysfunction and neurodegeneration in the npc1-/- mouse.

5. apoE3[K146N/R147W] acts as a dominant negative apoE form that prevents remnant clearance and inhibits the biogenesis of HDL.

Review 6. ApoE and Aβ in Alzheimer's disease: accidental encounters or partners?

7. GM2/GD2 and GM3 gangliosides have no effect on cellular cholesterol pools or turnover in normal or NPC1 mice.

8. Apolipoprotein E synthesis in human kidney, adrenal gland, and liver.

9. Low density lipoprotein degradation by mononuclear cells from normal and dyslipoproteinemic subjects.

10. Roles of apolipoproteins B and E in the cellular binding of very low density lipoproteins.