Literature DB >> 20584071

Role of glucocorticoid receptor polymorphism in adrenal incidentalomas.

Valentina Morelli1, Francesca Donadio, Cristina Eller-Vainicher, Valentina Cirello, Luca Olgiati, Chiara Savoca, Elisa Cairoli, Antonio Stefano Salcuni, Paolo Beck-Peccoz, Iacopo Chiodini.   

Abstract

BACKGROUND: Adrenal incidentalomas (AI) have been associated with and an increased prevalence of metabolic and bone complications. The N363S and BclI polymorphisms of the glucocorticoid receptor (GR) have been associated with an increased sensitivity to glucocorticoid (GC). This observational study aims to evaluate whether BclI and N363S polymorphisms play a role in the development of complications in AI.
MATERIALS AND METHODS: We enrolled 100 patients with AI (66 F; 34M). The presence of diabetes, arterial hypertension (AH), dyslipidaemia, osteoporosis and vertebral fracture (Fx), waist circumference and the Body Mass Index (BMI) were assessed. DNA samples were genotyped. Patients with wild-type BclI, wild-type N363S and heterozygous BclI polymorphism were classified as carriers of haplotype 1 (H1; n = 86), patients with homozygous BclI and heterozygous N363S polymorphism of GR of haplotype 2 (H2; n = 14).
RESULTS: We found no clinical or biochemical differences between haplotype 1 and 2 groups, but a higher prevalence of the simultaneous presence of Fx plus AH in H2 patients (H2 n = 7, H1 n = 16, P = 0.01). Logistic regression analysis showed that the presence of Fx and of AH and the combination of the presence of Fx plus AH were associated with the H2 genotype regardless of the degree of cortisol secretion, age, BMI and BMD (OR 4.88, 95%CI 1.47-18.40, P = 0.05; OR 8.25, 95%CI 0.98-69.52, P = 0.05; OR 7.25, 95%CI 1.57-35.78, P = 0.011; respectively).
CONCLUSIONS: In AI patients, the presence of the haplotype 2 of BclI and N363S is associated with the presence of AH, Fx and with the combination of Fx and AH.

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Year:  2010        PMID: 20584071     DOI: 10.1111/j.1365-2362.2010.02330.x

Source DB:  PubMed          Journal:  Eur J Clin Invest        ISSN: 0014-2972            Impact factor:   4.686


  20 in total

1.  Bone complications in patients with Cushing's syndrome: looking for clinical, biochemical, and genetic determinants.

Authors:  L Trementino; G Appolloni; L Ceccoli; G Marcelli; C Concettoni; M Boscaro; G Arnaldi
Journal:  Osteoporos Int       Date:  2013-10-15       Impact factor: 4.507

2.  Clinical and biochemical impact of BCL1 polymorphic genotype of the glucocorticoid receptor gene in patients with adrenal incidentalomas.

Authors:  M Tzanela; E Mantzou; K Saltiki; M Tampourlou; N Kalogeris; D Hadjidakis; S Tsagarakis; M Alevizaki
Journal:  J Endocrinol Invest       Date:  2011-07-05       Impact factor: 4.256

3.  The effects of cortisol and adrenal androgen on bone mass in Asians with and without subclinical hypercortisolism.

Authors:  S H Ahn; J H Kim; Y Y Cho; S Suh; B-J Kim; S Hong; S H Lee; J-M Koh; K-H Song
Journal:  Osteoporos Int       Date:  2019-02-04       Impact factor: 4.507

Review 4.  Autonomous cortisol secretion in adrenal incidentalomas.

Authors:  Marta Araujo-Castro; Miguel Antonio Sampedro Núñez; Mónica Marazuela
Journal:  Endocrine       Date:  2019-03-07       Impact factor: 3.633

5.  The degree of urinary hypercortisolism is not correlated with the severity of cushing's syndrome.

Authors:  Valentina Guarnotta; Marco C Amato; Rosario Pivonello; Giorgio Arnaldi; Alessandro Ciresi; Laura Trementino; Roberto Citarrella; Davide Iacuaniello; Grazia Michetti; Chiara Simeoli; Annamaria Colao; Carla Giordano
Journal:  Endocrine       Date:  2016-03-10       Impact factor: 3.633

6.  The association of cortisol and adrenal androgen with trabecular bone score in patients with adrenal incidentaloma with and without autonomous cortisol secretion.

Authors:  B-J Kim; M K Kwak; S H Ahn; J S Kim; S H Lee; J-M Koh
Journal:  Osteoporos Int       Date:  2018-07-03       Impact factor: 4.507

Review 7.  Adrenocortical incidentalomas and bone: from molecular insights to clinical perspectives.

Authors:  Barbara Altieri; Giovanna Muscogiuri; Stavroula A Paschou; Andromachi Vryonidou; Silvia Della Casa; Alfredo Pontecorvi; Martin Fassnacht; Cristina L Ronchi; John Newell-Price
Journal:  Endocrine       Date:  2018-08-02       Impact factor: 3.633

8.  Alteration in skeletal muscle mass in women with subclinical hypercortisolism.

Authors:  Jae Hyeon Kim; Mi Kyung Kwak; Seong Hee Ahn; Hyeonmok Kim; Yoon Young Cho; Sunghwan Suh; Beom-Jun Kim; Kee-Ho Song; Seung Hun Lee; Jung-Min Koh
Journal:  Endocrine       Date:  2018-05-01       Impact factor: 3.633

9.  Glucocorticoid receptor and molecular chaperones in the pathogenesis of adrenal incidentalomas: potential role of reduced sensitivity to glucocorticoids.

Authors:  Svetozar S Damjanovic; Jadranka A Antic; Bojana B Ilic; Bojana Beleslin Cokic; Miomira Ivovic; Sanja I Ognjanovic; Tatjana V Isailovic; Bojana M Popovic; Ivana B Bozic; Svetislav Tatic; Gordana Matic; Vera N Todorovic; Ivan Paunovic
Journal:  Mol Med       Date:  2013-01-22       Impact factor: 6.354

10.  Maximum adenoma diameter, regardless of uni- or bilaterality, is a risk factor for autonomous cortisol secretion in adrenal incidentalomas.

Authors:  M Araujo-Castro; C Robles Lázaro; P Parra Ramírez; R García Centeno; P Gracia Gimeno; M T Fernández-Ladreda; M A Sampedro Núñez; M Marazuela; H F Escobar-Morreale; P Valderrabano
Journal:  J Endocrinol Invest       Date:  2021-03-08       Impact factor: 4.256

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