Literature DB >> 20536609

Linkage of large-vessel carotid atherosclerotic stroke to inflammatory genes via a systematic screen.

Inna Belfer1, Tianxia Wu, Heather Hipp, Joan Walter, Michele Scully, Paul A Nyquist, Antonella Bollettino, David Goldman, Mitchell B Max, Thomas J DeGraba.   

Abstract

BACKGROUND: Inflammatory cytokines including the IL-1 family, TNF-alpha and IL-6 mediate the formation of thrombosis on the luminal surface of atherosclerotic plaques. Gene polymorphisms that regulate these cytokines' expression may explain part of the variation in susceptibility to stroke in patients with carotid atherosclerosis. The aim of this study was to evaluate the role of single-nucleotide polymorphisms (SNPs) and haplotypes in inflammatory genes as they relate to symptomatic carotid atherosclerosis.
METHODS: The study included 95 subjects with symptomatic (transient ischaemic attacks or stroke) and 113 subjects with asymptomatic carotid atherosclerotic disease. A panel of evenly spaced SNPs including previously reported functionally significant polymorphisms were genotyped for IL-1beta (10 SNPs), IL-1alpha (nine SNPs), IL-1RN (11 SNPs), IL-6 (seven SNPs) and TNF-alpha and TNF-beta (seven SNPs).
RESULTS: Using single SNP analysis, IL-1RN rs315934 (P=0.025), IL-1RN rs315946 (P=0.042), IL-1RN rs315921 (P=0.035), IL-6 rs1180243 (P=0.018) and IL-1alpha rs2071373 (P=0.025) were associated with decreased odds of symptomatic carotid disease. Additionally, two diplotypes of the IL-1RN gene (P=0.023 and 0.0064) and one diplotype in the IL-1alpha gene (P=0.02) were associated with a protective affect from cerebral ischaemic events. Logistic analysis for interaction of the protective SNPs reveals an additive effect of all SNP pair combinations.
CONCLUSION: These results suggest that genetic polymorphisms in proinflammatory genes may contribute to interindividual differences in the development of symptomatic carotid atherosclerotic disease.

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Year:  2010        PMID: 20536609      PMCID: PMC3437928          DOI: 10.1111/j.1747-4949.2010.00422.x

Source DB:  PubMed          Journal:  Int J Stroke        ISSN: 1747-4930            Impact factor:   5.266


  34 in total

1.  A study of twins and stroke.

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2.  Interleukin 1 induces endothelial cell procoagulant while suppressing cell-surface anticoagulant activity.

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6.  Beneficial effect of carotid endarterectomy in symptomatic patients with high-grade carotid stenosis.

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9.  Polymorphisms of the interleukin-1beta gene affect the risk of myocardial infarction and ischemic stroke at young age and the response of mononuclear cells to stimulation in vitro.

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  8 in total

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2.  Polygenic Risk for Depression Increases Risk of Ischemic Stroke: From the Stroke Genetics Network Study.

Authors:  Sylvia Wassertheil-Smoller; Qibin Qi; Tushar Dave; Braxton D Mitchell; Rebecca D Jackson; Simin Liu; Ki Park; Joel Salinas; Erin C Dunn; Enrique C Leira; Huichun Xu; Kathleen Ryan; Jordan W Smoller
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Review 3.  Inflammatory risk factors, biomarkers and associated therapy in ischaemic stroke.

Authors:  Charles C Esenwa; Mitchell S Elkind
Journal:  Nat Rev Neurol       Date:  2016-09-12       Impact factor: 42.937

4.  Polymorphisms in the TNFA and IL6 genes represent risk factors for autoimmune thyroid disease.

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Journal:  PLoS One       Date:  2014-08-15       Impact factor: 3.240

5.  Biomarker discovery in serum from patients with carotid atherosclerosis.

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6.  Genetic variants in IL1A and IL1B contribute to the susceptibility to 2009 pandemic H1N1 influenza A virus.

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Review 8.  Application of Single-Nucleotide Polymorphism-Related Risk Estimates in Identification of Increased Genetic Susceptibility to Cardiovascular Diseases: A Literature Review.

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  8 in total

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