Literature DB >> 20495373

MAP kinase phosphorylation is dispensable for cell division, but required for cell growth in Drosophila.

Neena Majumdar1, Gerardo L Paez, Shivangi M Inamdar, Mitchell D'Rozario, Daniel R Marenda.   

Abstract

Proper activation of the Ras/MAPK pathway is broadly required during development, and in many cases, signal transduction downstream of the receptor is linear. Thus, different mechanisms exist to properly regulate the large number of specific developmental outputs that are required by the activation of this pathway. Previously, we have reported a regulated cytoplasmic sequestration of phosphorylated MAPK (pMAPK) in developing Drosophila compound eyes and wings "called MAPK Cytoplasmic Hold". In the developing wing, we have shown that cytoplasmic hold promotes the differentiation of wing vein tissue, while pMAPK nuclear translocation regulates growth and division. We had also suggested that the Ras pathway signals for inducing cell growth and cell division split upstream of the nuclear translocation of MAPK itself. Here, we further refine the role of MAPK in Drosophila. We report evidence that suggests, for the first time, that the phosphorylation of MAPK is itself another step in the regulation of cell growth and division in both Drosophila wing and eye cells. We show that inhibition of MAPK phosphorylation, or pMAPK nuclear translocation, is sufficient to block cell growth, but not cell division. These data suggest that non-phosphorylated MAPK is sufficient to induce cell division, but not cell growth, once inside the nucleus of the cell.

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Year:  2010        PMID: 20495373      PMCID: PMC3322500          DOI: 10.4161/fly.4.3.12001

Source DB:  PubMed          Journal:  Fly (Austin)        ISSN: 1933-6934            Impact factor:   2.160


  46 in total

1.  Ras1 promotes cellular growth in the Drosophila wing.

Authors:  D A Prober; B A Edgar
Journal:  Cell       Date:  2000-02-18       Impact factor: 41.582

Review 2.  Dimerization in MAP-kinase signaling.

Authors:  M H Cobb; E J Goldsmith
Journal:  Trends Biochem Sci       Date:  2000-01       Impact factor: 13.807

Review 3.  Receptor tyrosine kinases: specific outcomes from general signals.

Authors:  M A Simon
Journal:  Cell       Date:  2000-09-29       Impact factor: 41.582

4.  The Drosophila SNR1 (SNF5/INI1) subunit directs essential developmental functions of the Brahma chromatin remodeling complex.

Authors:  Daniel R Marenda; Claudia B Zraly; Yun Feng; Susan Egan; Andrew K Dingwall
Journal:  Mol Cell Biol       Date:  2003-01       Impact factor: 4.272

5.  Cell survival promoted by the Ras-MAPK signaling pathway by transcription-dependent and -independent mechanisms.

Authors:  A Bonni; A Brunet; A E West; S R Datta; M A Takasu; M E Greenberg
Journal:  Science       Date:  1999-11-12       Impact factor: 47.728

6.  Drosophila Cdk4 is required for normal growth and is dispensable for cell cycle progression.

Authors:  C A Meyer; H W Jacobs; S A Datar; W Du; B A Edgar; C F Lehner
Journal:  EMBO J       Date:  2000-09-01       Impact factor: 11.598

7.  Evidence for existence of a nuclear pore complex-mediated, cytosol-independent pathway of nuclear translocation of ERK MAP kinase in permeabilized cells.

Authors:  Y Matsubayashi; M Fukuda; E Nishida
Journal:  J Biol Chem       Date:  2001-09-06       Impact factor: 5.157

Review 8.  Signaling by the Drosophila epidermal growth factor receptor pathway during development.

Authors:  Ben Zion Shilo
Journal:  Exp Cell Res       Date:  2003-03-10       Impact factor: 3.905

9.  Nuclear import of activated D-ERK by DIM-7, an importin family member encoded by the gene moleskin.

Authors:  J A Lorenzen; S E Baker; F Denhez; M B Melnick; D L Brower; L A Perkins
Journal:  Development       Date:  2001-04       Impact factor: 6.868

10.  Analysis of Drosophila photoreceptor axon guidance in eye-specific mosaics.

Authors:  T P Newsome; B Asling; B J Dickson
Journal:  Development       Date:  2000-02       Impact factor: 6.868

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