Literature DB >> 20466981

Cardioprotection of ischemia/reperfusion injury by cholesterol-dependent MG53-mediated membrane repair.

Xianhua Wang1, Wenjun Xie, Yi Zhang, Peihui Lin, Liang Han, Peidong Han, Yanru Wang, Zheng Chen, Guangju Ji, Ming Zheng, Noah Weisleder, Rui-Ping Xiao, Hiroshi Takeshima, Jianjie Ma, Heping Cheng.   

Abstract

RATIONALE: Unrepaired cardiomyocyte membrane injury causes irreplaceable cell loss, leading to myocardial fibrosis and eventually heart failure. However, the cellular and molecular mechanisms of cardiac membrane repair are largely unknown. MG53, a newly identified striated muscle-specific protein, is involved in skeletal muscle membrane repair. But the role of MG53 in the heart has not been determined.
OBJECTIVE: We sought to investigate whether MG53 mediates membrane repair in cardiomyocytes and, if so, the cellular and molecular mechanism underlying MG53-mediated membrane repair in cardiomyocytes. Moreover, we determined possible cardioprotective effect of MG53-mediated membrane repair. METHODS AND
RESULTS: We demonstrated that MG53 is crucial to the emergency membrane repair response in cardiomyocytes and protects the heart from stress-induced loss of cardiomyocytes. Disruption of the sarcolemmal membrane by mechanical, electric, chemical, or metabolic insults caused rapid and robust translocation of MG53 toward the injury sites. Ablation of MG53 prevented sarcolemmal resealing after infrared laser-induced membrane damage in intact heart, and exacerbated mitochondrial dysfunction and loss of cardiomyocytes during ischemia/reperfusion injury. Unexpectedly, the MG53-mediated cardiac membrane repair was mediated by a cholesterol-dependent mechanism: depletion of membrane cholesterol abolished, and its recovery restored injury-induced membrane translocation of MG53. The redox status of MG53 did not affect initiation of MG53 translocation, whereas MG53 oxidation conferred stability to the membrane repair patch.
CONCLUSIONS: Thus, cholesterol-dependent MG53-mediated membrane repair is a vital, heretofore unappreciated cardioprotective mechanism against a multitude of insults and may bear important therapeutic implications.

Entities:  

Mesh:

Substances:

Year:  2010        PMID: 20466981     DOI: 10.1161/CIRCRESAHA.109.215822

Source DB:  PubMed          Journal:  Circ Res        ISSN: 0009-7330            Impact factor:   17.367


  82 in total

1.  Nonmuscle myosin IIA facilitates vesicle trafficking for MG53-mediated cell membrane repair.

Authors:  Peihui Lin; Hua Zhu; Chuanxi Cai; Xianhua Wang; Chunmei Cao; Ruiping Xiao; Zui Pan; Noah Weisleder; Hiroshi Takeshima; Jianjie Ma
Journal:  FASEB J       Date:  2012-01-17       Impact factor: 5.191

2.  Recombinant MG53 protein modulates therapeutic cell membrane repair in treatment of muscular dystrophy.

Authors:  Noah Weisleder; Norio Takizawa; Peihui Lin; Xianhua Wang; Chunmei Cao; Yan Zhang; Tao Tan; Christopher Ferrante; Hua Zhu; Pin-Jung Chen; Rosalie Yan; Matthew Sterling; Xiaoli Zhao; Moonsun Hwang; Miyuki Takeshima; Chuanxi Cai; Heping Cheng; Hiroshi Takeshima; Rui-Ping Xiao; Jianjie Ma
Journal:  Sci Transl Med       Date:  2012-06-20       Impact factor: 17.956

Review 3.  Plasma Membrane Repair: A Central Process for Maintaining Cellular Homeostasis.

Authors:  Alisa D Blazek; Brian J Paleo; Noah Weisleder
Journal:  Physiology (Bethesda)       Date:  2015-11

Review 4.  Membrane Repair: Mechanisms and Pathophysiology.

Authors:  Sandra T Cooper; Paul L McNeil
Journal:  Physiol Rev       Date:  2015-10       Impact factor: 37.312

5.  The cardiac syndecan-4 interactome reveals a role for syndecan-4 in nuclear translocation of muscle LIM protein (MLP).

Authors:  Sabrina Bech Mathiesen; Marianne Lunde; Jan Magnus Aronsen; Andreas Romaine; Anita Kaupang; Marita Martinsen; Gustavo Antonio de Souza; Tuula A Nyman; Ivar Sjaastad; Geir Christensen; Cathrine Rein Carlson
Journal:  J Biol Chem       Date:  2019-04-09       Impact factor: 5.157

6.  Targeted disruption of PDE3B, but not PDE3A, protects murine heart from ischemia/reperfusion injury.

Authors:  Youn Wook Chung; Claudia Lagranha; Yong Chen; Junhui Sun; Guang Tong; Steven C Hockman; Faiyaz Ahmad; Shervin G Esfahani; Dahae H Bae; Nazari Polidovitch; Jian Wu; Dong Keun Rhee; Beom Seob Lee; Marjan Gucek; Mathew P Daniels; Christine A Brantner; Peter H Backx; Elizabeth Murphy; Vincent C Manganiello
Journal:  Proc Natl Acad Sci U S A       Date:  2015-04-15       Impact factor: 11.205

7.  MG53 participates in ischaemic postconditioning through the RISK signalling pathway.

Authors:  Yan Zhang; Fengxiang Lv; Li Jin; Wei Peng; Ruisheng Song; Jianjie Ma; Chun-Mei Cao; Rui-Ping Xiao
Journal:  Cardiovasc Res       Date:  2011-02-01       Impact factor: 10.787

8.  Expression levels of sarcolemmal membrane repair proteins following prolonged exercise training in mice.

Authors:  Jenna Alloush; Steve R Roof; Eric X Beck; Mark T Ziolo; Noah Weisleder
Journal:  Indian J Biochem Biophys       Date:  2013-10       Impact factor: 1.918

Review 9.  Trimeric intracellular cation channels and sarcoplasmic/endoplasmic reticulum calcium homeostasis.

Authors:  Xinyu Zhou; Peihui Lin; Daiju Yamazaki; Ki Ho Park; Shinji Komazaki; S R Wayne Chen; Hiroshi Takeshima; Jianjie Ma
Journal:  Circ Res       Date:  2014-02-14       Impact factor: 17.367

Review 10.  Poloxamer 188 (p188) as a membrane resealing reagent in biomedical applications.

Authors:  Joseph G Moloughney; Noah Weisleder
Journal:  Recent Pat Biotechnol       Date:  2012-12
View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.