Literature DB >> 20394945

Infantile hypercalcemia and hypercalciuria: new insights into a vitamin D-dependent mechanism and response to ketoconazole treatment.

Minh Nguyen1, Henri Boutignon, Eric Mallet, Agnes Linglart, Huguette Guillozo, Frederic Jehan, Michele Garabedian.   

Abstract

OBJECTIVE: To analyze vitamin D metabolism and response to ketoconazole, an imidazole derivative that inhibits the vitamin D-1-hydroxylase, in infants with idiopathic hypercalcemia, and hypercalciuria. STUDY
DESIGN: Twenty infants (4 days-17 months) with hypercalcemia, severe hypercalciuria, and low parathyroid hormone level, (10 had nephrocalcinosis), including 10 treated with ketoconazole (3-9 mg/kg/day), were followed to the age of 2 to 51 months. Vitamin D receptor expression (VDR), 24-hydroxylase activity, and functional gene polymorphisms of vitamin D metabolism regulators VDR(rs4516035), 1-hydroxylase(rs10877012), 24-hydroxylase(rs2248359), FGF23(rs7955866), Klotho(rs9536314, rs564481, rs648202), were evaluated.
RESULTS: Serum calcium levels, which occurred faster in the ketoconazole group (0.7 +/- 0.2 versus 2.4 +/- 0.6 months; P = .0076), and urinary calcium excretion (2.5 +/- 0.5 versus 4.2 +/- 1.7 months) normalized in all patients. Serum 1,25-(OH)2D levels were high normal and positively correlated to 25-(OH)D levels. Serum 24,25-(OH)2D levels were low normal, and skin fibroblasts from 1 patient showed defective up-regulation of the 24-hydroxylase by 1,25-(OH)2D despite normal VDR binding ability. An abnormally low prevalence of haplotype CC/CC for H589H/A749A in Klotho gene was found in patients and family members.
CONCLUSIONS: Ketoconazole is a potentially useful and safe agent for treatment of infantile hypercalcemia. Abnormal vitamin D metabolism is suggested as the mechanism, possibly involving defective up-regulation of the 24-hydroxylase by 1,25-(OH)2D3, and the klotho-FGF23 axis. Copyright (c) 2010 Mosby, Inc. All rights reserved.

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Year:  2010        PMID: 20394945     DOI: 10.1016/j.jpeds.2010.02.025

Source DB:  PubMed          Journal:  J Pediatr        ISSN: 0022-3476            Impact factor:   4.406


  19 in total

1.  Take another CYP: confirming a novel mechanism for "idiopathic" hypercalcemia.

Authors:  Thomas O Carpenter
Journal:  J Clin Endocrinol Metab       Date:  2012-03       Impact factor: 5.958

2.  Hyperphosphatemic tumoral calcinosis caused by FGF23 compound heterozygous mutations: what are the therapeutic options for a better control of phosphatemia?

Authors:  Debora Claramunt-Taberner; Aurélia Bertholet-Thomas; Marie-Christine Carlier; Frédérique Dijoud; Franck Chotel; Caroline Silve; Justine Bacchetta
Journal:  Pediatr Nephrol       Date:  2018-03-28       Impact factor: 3.714

3.  Genetic defect in CYP24A1, the vitamin D 24-hydroxylase gene, in a patient with severe infantile hypercalcemia.

Authors:  Andrew Dauber; Thutrang T Nguyen; Etienne Sochett; David E C Cole; Ronald Horst; Steven A Abrams; Thomas O Carpenter; Joel N Hirschhorn
Journal:  J Clin Endocrinol Metab       Date:  2011-11-23       Impact factor: 5.958

Review 4.  Hypercalcemia: a consultant's approach.

Authors:  Ari Auron; Uri S Alon
Journal:  Pediatr Nephrol       Date:  2017-09-06       Impact factor: 3.714

5.  1,25-(OH)2D-24 Hydroxylase (CYP24A1) Deficiency as a Cause of Nephrolithiasis.

Authors:  Galina Nesterova; May Christine Malicdan; Kaori Yasuda; Toshiyuki Sakaki; Thierry Vilboux; Carla Ciccone; Ronald Horst; Yan Huang; Gretchen Golas; Wendy Introne; Marjan Huizing; David Adams; Cornelius F Boerkoel; Michael T Collins; William A Gahl
Journal:  Clin J Am Soc Nephrol       Date:  2013-01-04       Impact factor: 8.237

6.  25-Hydroxyvitamin D Can Interfere With a Common Assay for 1,25-Dihydroxyvitamin D in Vitamin D Intoxication.

Authors:  Colin P Hawkes; Sarah Schnellbacher; Ravinder J Singh; Michael A Levine
Journal:  J Clin Endocrinol Metab       Date:  2015-06-29       Impact factor: 5.958

7.  CYP3A4 Induction by Rifampin: An Alternative Pathway for Vitamin D Inactivation in Patients With CYP24A1 Mutations.

Authors:  Colin Patrick Hawkes; Dong Li; Hakon Hakonarson; Kevin E Meyers; Kenneth E Thummel; Michael A Levine
Journal:  J Clin Endocrinol Metab       Date:  2017-05-01       Impact factor: 5.958

8.  Sotos syndrome, infantile hypercalcemia, and nephrocalcinosis: a contiguous gene syndrome.

Authors:  Joanna Kenny; Melissa M Lees; Susan Drury; Angela Barnicoat; William Van't Hoff; Rodger Palmer; Deborah Morrogh; Jonathan J Waters; Nicholas J Lench; Detlef Bockenhauer
Journal:  Pediatr Nephrol       Date:  2011-05-20       Impact factor: 3.714

Review 9.  A lifetime of hypercalcemia and hypercalciuria, finally explained.

Authors:  Thomas P Jacobs; Martin Kaufman; Glenville Jones; Rajiv Kumar; Karl-Peter Schlingmann; Sue Shapses; John P Bilezikian
Journal:  J Clin Endocrinol Metab       Date:  2014-01-13       Impact factor: 5.958

10.  Mild Idiopathic Infantile Hypercalcemia-Part 2: A Longitudinal Observational Study.

Authors:  Nina Lenherr-Taube; Michelle Furman; Esther Assor; Yesmino Elia; Carol Collins; Kenneth Thummel; Michael A Levine; Etienne Sochett
Journal:  J Clin Endocrinol Metab       Date:  2021-09-27       Impact factor: 6.134

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