Literature DB >> 20362716

The relationship between coronary stenosis severity and compression type coronary artery movement in acute myocardial infarction.

Kim H Chan1, Chirapan Chawantanpipat, Tim Gattorna, Thamarath Chantadansuwan, Adrienne Kirby, Ann Madden, Anthony Keech, Martin K C Ng.   

Abstract

BACKGROUND: Acute myocardial infarction is thought to occur at sites of minor coronary stenosis. Recent data challenge this and also propose a role for coronary artery movement (CAM) in plaque instability. We examined the relationship between coronary stenosis severity, CAM pattern, and infarct-related lesions (IRLs) in acute myocardial infarction.
METHODS: We investigated 203 consecutive patients with ST-segment elevation myocardial infarction after successful fibrinolysis. Quantitative coronary angiography, CAM pattern, and extent score (atheroma burden) analysis was performed for each coronary artery segment.
RESULTS: The IRL stenosis was at least moderate (>50%) and severe (>70%) in 78% and 31% of patients, respectively. Culprit arteries were associated with higher atheroma extent scores (25.2 vs 21.6, P < .001). Analysis of 2,228 coronary segments showed that stenosis severity and IRLs were highly correlated, such that the likelihood of being a culprit segment progressively increased with worsening stenosis (odds ratio [OR] 30.0, 95% confidence interval [CI] 19.0-47.6, P < .001, for >70% vs <30% stenosis). Compression CAM was also strongly associated with culprit segments (OR 3.4, 95% CI 2.6-4.5, P < .001). In addition, compression CAM and stenosis severity were strongly correlated, with the likelihood of a coronary segment having compression CAM progressively increasing with worsening stenosis (OR 56.4, 95% CI 37.9-83.8, P < .001, for >70% vs <30% stenosis).
CONCLUSIONS: In patients with ST-segment elevation myocardial infarction, there is a strong relationship between stenosis severity and IRLs. Our study also raises the hypothesis that compression CAM may accelerate atherosclerosis and predispose to plaque vulnerability. Copyright 2010 Mosby, Inc. All rights reserved.

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Year:  2010        PMID: 20362716     DOI: 10.1016/j.ahj.2009.12.036

Source DB:  PubMed          Journal:  Am Heart J        ISSN: 0002-8703            Impact factor:   4.749


  4 in total

1.  High-grade culprit lesions are a common cause of ST-segment elevation myocardial infarction.

Authors:  Michael Liang; Takashi Kajiya; Mark Y Chan; Edgar Tay; Chi-Hang Lee; Arthur Mark Richards; Adrian F Low; Huay Cheem Tan
Journal:  Singapore Med J       Date:  2015-06       Impact factor: 1.858

2.  The Relationship between Coronary Artery Movement Type andStenosis Severity with Acute Myocardial Infarction.

Authors:  Samad Ghaffari; Siamak Erfanparast; Ahmad Separham; Sepideh Sokhanvar; Mehrdad Yavarikia; Leili Pourafkari
Journal:  J Cardiovasc Thorac Res       Date:  2013-06-27

Review 3.  Multi-Modality Imaging for Prevention of Coronary Artery Disease and Myocardial Infarction in the General Population: Ready for Prime Time?

Authors:  Daan Ties; Paulien van Dorp; Gabija Pundziute; Erik Lipsic; Carlijn M van der Aalst; Matthijs Oudkerk; Harry J de Koning; Rozemarijn Vliegenthart; Pim van der Harst
Journal:  J Clin Med       Date:  2022-05-24       Impact factor: 4.964

4.  The compression type of coronary artery motion in patients with ST-segment elevation acute myocardial infarction and normal controls: a case-control study.

Authors:  Aiden Jc O'Loughlin; Karen Byth; John K French; David Ab Richards; Annemarie Hennessy; A Robert Denniss; Pramesh Kovoor
Journal:  BMC Res Notes       Date:  2011-03-07
  4 in total

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