Literature DB >> 20331791

Pneumococcal modification of host sugars: a major contributor to colonization of the human airway?

S J King1.   

Abstract

Streptococcus pneumoniae colonization of the human airway is an essential precursor for disease; however, the mechanisms by which the bacterium establishes and maintains colonization are poorly understood. It is becoming increasingly clear that S. pneumoniae expresses glycosidases that can modify many glycan structures present in the human airway, including N-linked glycans, O-linked glycans, and glycosaminoglycans. Many of these glycosidases have been shown to contribute to in vivo colonization. Although the precise role of these glycosidases during colonization remains to be elucidated, in vitro assays suggest that pneumococcal modification of host sugars may contribute to colonization in a variety of ways. Experimental evidence supports a role for pneumococcal glycosidases in providing a carbon source for growth, biofilm formation, competition with other bacteria within the airway, and exposing receptors for adherence. Herein we review the ability of S. pneumoniae to modify host sugars and the functional effects of these modifications.

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Year:  2010        PMID: 20331791     DOI: 10.1111/j.2041-1014.2009.00564.x

Source DB:  PubMed          Journal:  Mol Oral Microbiol        ISSN: 2041-1006            Impact factor:   3.563


  60 in total

Review 1.  Host Sialic Acids: A Delicacy for the Pathogen with Discerning Taste.

Authors:  Brandy L Haines-Menges; W Brian Whitaker; J B Lubin; E Fidelma Boyd
Journal:  Microbiol Spectr       Date:  2015-08

2.  Sialic acid-mediated gene expression in Streptococcus pneumoniae and role of NanR as a transcriptional activator of the nan gene cluster.

Authors:  Muhammad Afzal; Sulman Shafeeq; Hifza Ahmed; Oscar P Kuipers
Journal:  Appl Environ Microbiol       Date:  2015-02-27       Impact factor: 4.792

3.  Sialic acid transport contributes to pneumococcal colonization.

Authors:  Carolyn Marion; Amanda M Burnaugh; Shireen A Woodiga; Samantha J King
Journal:  Infect Immun       Date:  2010-12-28       Impact factor: 3.441

4.  Free Sialic Acid Acts as a Signal That Promotes Streptococcus pneumoniae Invasion of Nasal Tissue and Nonhematogenous Invasion of the Central Nervous System.

Authors:  Brandon L Hatcher; Joanetha Y Hale; David E Briles
Journal:  Infect Immun       Date:  2016-08-19       Impact factor: 3.441

5.  LacR is a repressor of lacABCD and LacT is an activator of lacTFEG, constituting the lac gene cluster in Streptococcus pneumoniae.

Authors:  Muhammad Afzal; Sulman Shafeeq; Oscar P Kuipers
Journal:  Appl Environ Microbiol       Date:  2014-06-20       Impact factor: 4.792

6.  Absence of capsule reveals glycan-mediated binding and recognition of salivary mucin MUC7 by Streptococcus pneumoniae.

Authors:  S Thamadilok; H Roche-Håkansson; A P Håkansson; S Ruhl
Journal:  Mol Oral Microbiol       Date:  2015-08-17       Impact factor: 3.563

7.  Induction of a quorum sensing pathway by environmental signals enhances group A streptococcal resistance to lysozyme.

Authors:  Jennifer C Chang; Juan Cristobal Jimenez; Michael J Federle
Journal:  Mol Microbiol       Date:  2015-07-17       Impact factor: 3.501

Review 8.  Composition and development of oral bacterial communities.

Authors:  Robert J Palmer
Journal:  Periodontol 2000       Date:  2014-02       Impact factor: 7.589

9.  Pneumococcal Neuraminidase Substrates Identified through Comparative Proteomics Enabled by Chemoselective Labeling.

Authors:  Janet E McCombs; Jennifer J Kohler
Journal:  Bioconjug Chem       Date:  2016-03-22       Impact factor: 4.774

10.  Modulation of expression in BEAS-2B airway epithelial cells of α-L-fucosidase A1 and A2 by Th1 and Th2 cytokines, and overexpression of α-L-fucosidase 2.

Authors:  Anna D Sobkowicz; Mary E Gallagher; Colm J Reid; Daniel Crean; Stephen D Carrington; Jane A Irwin
Journal:  Mol Cell Biochem       Date:  2014-01-28       Impact factor: 3.396

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