Literature DB >> 20300059

Genetic studies of human DNA repair proteins using yeast as a model system.

Monika Aggarwal1, Robert M Brosh.   

Abstract

Understanding the roles of human DNA repair proteins in genetic pathways is a formidable challenge to many researchers. Genetic studies in mammalian systems have been limited due to the lack of readily available tools including defined mutant genetic cell lines, regulatory expression systems, and appropriate selectable markers. To circumvent these difficulties, model genetic systems in lower eukaryotes have become an attractive choice for the study of functionally conserved DNA repair proteins and pathways. We have developed a model yeast system to study the poorly defined genetic functions of the Werner syndrome helicase-nuclease (WRN) in nucleic acid metabolism. Cellular phenotypes associated with defined genetic mutant backgrounds can be investigated to clarify the cellular and molecular functions of WRN through its catalytic activities and protein interactions. The human WRN gene and associated variants, cloned into DNA plasmids for expression in yeast, can be placed under the control of a regulatory plasmid element. The expression construct can then be transformed into the appropriate yeast mutant background, and genetic function assayed by a variety of methodologies. Using this approach, we determined that WRN, like its related RecQ family members BLM and Sgs1, operates in a Top3-dependent pathway that is likely to be important for genomic stability. This is described in our recent publication at www.impactaging.com. Detailed methods of specific assays for genetic complementation studies in yeast are provided in this paper.

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Year:  2010        PMID: 20300059      PMCID: PMC3168208          DOI: 10.3791/1639

Source DB:  PubMed          Journal:  J Vis Exp        ISSN: 1940-087X            Impact factor:   1.355


  6 in total

1.  The yeast type I topoisomerase Top3 interacts with Sgs1, a DNA helicase homolog: a potential eukaryotic reverse gyrase.

Authors:  S Gangloff; J P McDonald; C Bendixen; L Arthur; R Rothstein
Journal:  Mol Cell Biol       Date:  1994-12       Impact factor: 4.272

2.  WRN helicase defective in the premature aging disorder Werner syndrome genetically interacts with topoisomerase 3 and restores the top3 slow growth phenotype of sgs1 top3.

Authors:  Monika Aggarwal; Robert M Brosh
Journal:  Aging (Albany NY)       Date:  2009-02-05       Impact factor: 5.682

3.  In vivo function of the conserved non-catalytic domain of Werner syndrome helicase in DNA replication.

Authors:  Sudha Sharma; Joshua A Sommers; Robert M Brosh
Journal:  Hum Mol Genet       Date:  2004-07-28       Impact factor: 6.150

4.  Werner syndrome protein contains three structure-specific DNA binding domains.

Authors:  Cayetano von Kobbe; Nicolas H Thomä; Bryan K Czyzewski; Nikola P Pavletich; Vilhelm A Bohr
Journal:  J Biol Chem       Date:  2003-10-08       Impact factor: 5.157

5.  Mutations in homologous recombination genes rescue top3 slow growth in Saccharomyces cerevisiae.

Authors:  Erika Shor; Serge Gangloff; Marisa Wagner; Justin Weinstein; Gavrielle Price; Rodney Rothstein
Journal:  Genetics       Date:  2002-10       Impact factor: 4.562

6.  Studies on the transformation of intact yeast cells by the LiAc/SS-DNA/PEG procedure.

Authors:  R D Gietz; R H Schiestl; A R Willems; R A Woods
Journal:  Yeast       Date:  1995-04-15       Impact factor: 3.239

  6 in total
  1 in total

Review 1.  Functional analyses of human DNA repair proteins important for aging and genomic stability using yeast genetics.

Authors:  Monika Aggarwal; Robert M Brosh
Journal:  DNA Repair (Amst)       Date:  2012-02-18
  1 in total

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