Literature DB >> 20298382

Regular use of non-steroidal anti-inflammatory drugs increases the risk of adult-onset asthma: a population-based follow-up study.

Simon Francis Thomsen1, Kirsten Ohm Kyvik, Lars Rauff Skadhauge, Ida Steffensen, Vibeke Backer.   

Abstract

BACKGROUND: Little is known about the relation between regular use of non-steroidal anti-inflammatory drugs (NSAIDs) and the risk of asthma at the population level. The aim of this study was to examine a possible association between intake of NSAIDs and risk of adult-onset asthma.
METHODS: Using data from two multidisciplinary postal questionnaire surveys concerning health and lifestyle, we prospectively studied 19 349 adult twins enrolled in the nationwide Danish Twin Registry.
RESULTS: We found a higher prevalence of new-onset asthma in subjects who used NSAIDs (other than aspirin) regularly compared with non-users (7.7% vs 4.3%), OR = 1.87 (1.25-2.81), P = 0.002. The result remained significant after adjusting for sex, age, smoking, BMI, hay fever, eczema and intake of medications other than NSAIDs, OR = 1.90 (1.26-2.85), P = 0.002.
CONCLUSIONS: Regular use of NSAIDs other than aspirin may be a risk factor for adult-onset asthma. This observation must be accommodated in explanations of the relationship between use of analgesics and risk of asthma.

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Year:  2009        PMID: 20298382     DOI: 10.1111/j.1752-699X.2008.00113.x

Source DB:  PubMed          Journal:  Clin Respir J        ISSN: 1752-6981            Impact factor:   2.570


  3 in total

1.  Prostaglandin I2 Signaling and Inhibition of Group 2 Innate Lymphoid Cell Responses.

Authors:  Weisong Zhou; Shinji Toki; Jian Zhang; Kasia Goleniewksa; Dawn C Newcomb; Jacqueline Y Cephus; Daniel E Dulek; Melissa H Bloodworth; Matthew T Stier; Vasiliy Polosuhkin; Rama D Gangula; Simon A Mallal; David H Broide; R Stokes Peebles
Journal:  Am J Respir Crit Care Med       Date:  2016-01-01       Impact factor: 21.405

2.  Cyclooxygenase inhibition abrogates aeroallergen-induced immune tolerance by suppressing prostaglandin I2 receptor signaling.

Authors:  Weisong Zhou; Kasia Goleniewska; Jian Zhang; Daniel E Dulek; Shinji Toki; Matthew T Lotz; Dawn C Newcomb; Madison G Boswell; Vasiliy V Polosukhin; Ginger L Milne; Pingsheng Wu; Martin L Moore; Garret A FitzGerald; R Stokes Peebles
Journal:  J Allergy Clin Immunol       Date:  2014-07-18       Impact factor: 10.793

3.  Prostaglandin E₂ suppresses allergic sensitization and lung inflammation by targeting the E prostanoid 2 receptor on T cells.

Authors:  Zbigniew Zasłona; Katsuhide Okunishi; Emilie Bourdonnay; Racquel Domingo-Gonzalez; Bethany B Moore; Nicholas W Lukacs; David M Aronoff; Marc Peters-Golden
Journal:  J Allergy Clin Immunol       Date:  2013-09-24       Impact factor: 10.793

  3 in total

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