Literature DB >> 20202060

The high rate of familial lichen sclerosus suggests a genetic contribution: an observational cohort study.

V Sherman1, T McPherson, M Baldo, A Salim, X H Gao, F Wojnarowska.   

Abstract

BACKGROUND: Familial lichen sclerosus (LS) has been described in only 37 families. We feel that the association is under-reported.
OBJECTIVES: To determine the percentage of patients with LS who have a positive family history.
METHOD: A large observational-cohort study of a total of 1052 females at vulval clinics within a University Hospital with a diagnosis of LS of the vulva (clinical diagnosis was confirmed in 80% of cases by histology). Patients were questioned as to family history of LS or balanitis xerotica obliterans; male circumcision for medical reasons; vulval cancer; and routine medical and family history. The outcome was the presence or absence of personal or family history of LS, autoimmune disorder or vulval cancer.
RESULTS: In total 1052 patients were investigated. Of these, 126 (12%) had a positive family history of LS. These patients belonged to 95 families. Vulval cancer was significantly increased in those with a family history of LS compared with those without (4.1% vs. 1.2%, P < 0.05). There was more associated autoimmune disease in familial LS than in sporadic LS, although this was not statistically significant. (7% vs. 5%, P > 0.2).
CONCLUSION: Our data from a large cohort of patients with LS provide evidence of an increased risk for family members to develop LS. This indicates a likely genetic component in the aetiology of LS.

Entities:  

Mesh:

Year:  2010        PMID: 20202060     DOI: 10.1111/j.1468-3083.2010.03572.x

Source DB:  PubMed          Journal:  J Eur Acad Dermatol Venereol        ISSN: 0926-9959            Impact factor:   6.166


  29 in total

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Authors:  Bradley A Erickson; Sean P Elliott; Jeremy B Myers; Bryan B Voelzke; Thomas G Smith; Christopher D McClung; Nejd F Alsikafi; Alex J Vanni; Will O Brant; Joshua A Broghammer; Christopher A Tam; Lee C Zhao; Jill C Buckley; Benjamin N Breyer
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