Literature DB >> 20194711

Do the actions of glucagon-like peptide-1 on gastric emptying, appetite, and food intake involve release of amylin in humans?

Meena Asmar1, Michael Bache, Filip K Knop, Sten Madsbad, Jens J Holst.   

Abstract

OBJECTIVE: Amylin, cosecreted with insulin, has like glucagon-like peptide-1 (GLP-1) been reported to inhibit glucagon secretion, delay gastric emptying, and reduce appetite and food intake. We investigated whether the effects of GLP-1 on gastric emptying, appetite, and food intake are mediated directly or indirectly via release of amylin.
DESIGN: Eleven C-peptide and amylin-negative patients with type 1 diabetes mellitus (T1DM) and 12 matched healthy controls participated in a placebo-controlled, randomized, single-blinded, crossover study. With glucose clamped between 6 and 9 mm, near-physiological infusions of GLP-1, human amylin, pramlintide, or saline were given for 270 min during and after a fixed meal. Gastric emptying was measured using paracetamol, appetite using visual analog scales, and food intake during a subsequent ad libitum meal (at 240 min).
RESULTS: In T1DM, gastric emptying, food intake, and appetite were reduced equally during low GLP-1 and amylin infusion compared with the saline infusion (P < 0.05). The controls showed stronger suppression of gastric emptying (P < 0.0001) and food intake (P < 0.01) with GLP-1 compared to amylin. Postprandial glucagon responses were reduced in controls and T1DM during GLP-1 and amylin infusions (P < 0.05). Amylin and pramlintide infusion had similar effects.
CONCLUSIONS: GLP-1 exerts its effect on gastric emptying, appetite, food intake, and glucagon secretion directly, although secretion of amylin may contribute to some of these effects in healthy control subjects.

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Year:  2010        PMID: 20194711     DOI: 10.1210/jc.2009-2133

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  12 in total

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Review 4.  Current Therapies That Modify Glucagon Secretion: What Is the Therapeutic Effect of Such Modifications?

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5.  Incretins and amylin: neuroendocrine communication between the gut, pancreas, and brain in control of food intake and blood glucose.

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Journal:  J Clin Endocrinol Metab       Date:  2016-03-01       Impact factor: 5.958

Review 7.  Glucose-dependent insulinotropic polypeptide and glucagon-like peptide-1: Incretin actions beyond the pancreas.

Authors:  Yutaka Seino; Daisuke Yabe
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Authors:  Sarah W Lai; Elaine de Heuvel; Laurie E Wallace; Bolette Hartmann; Jens J Holst; Mary E Brindle; Prasanth K Chelikani; David L Sigalet
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Review 9.  Optimal Pharmacologic Treatment Strategies in Obesity and Type 2 Diabetes.

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10.  Effects of solid-phase extraction of plasma in measuring gut metabolic hormones in fasted and fed blood of lean and diet-induced obese rats.

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Journal:  Physiol Rep       Date:  2016-05
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