Literature DB >> 20192952

Genetic variation in the indoleamine 2,3-dioxygenase gene in pre-eclampsia.

Haruki Nishizawa1, Takema Kato, Sayuri Ota, Sachie Nishiyama, Kanako Pryor-Koishi, Machiko Suzuki, Makiko Tsutsumi, Hidehito Inagaki, Hiroki Kurahashi, Yasuhiro Udagawa.   

Abstract

PROBLEM: To investigate the contribution of genomic variations in the indoleamine 2,3-dioxygenase (IDO) gene to the onset of pre-eclampsia. METHOD OF STUDY: We examined sequence variations in the IDO1 gene using placental genomic DNA from 35 pre-eclamptic patients and 32 normotensive pregnant women.
RESULTS: A case-control study revealed that none of the common variants influences the risk of disease. Sequencing of each IDO1 exon in diseased subjects revealed rare variants. This variation, c.-147_150delGAAA, was located within the 5'-untranslated region of the IDO1 gene, and its homozygote was identified only in pre-eclamptic subjects. However, despite the low levels of IDO expression and enzyme activity in the c.-147_150delGAAA homozygote, reporter assays indicated that this variation does not affect gene expression.
CONCLUSION: Our findings indicate that genetic alteration of fetal IDO gene does not appear to be a primary cause of pre-eclampsia.

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Year:  2010        PMID: 20192952     DOI: 10.1111/j.1600-0897.2010.00820.x

Source DB:  PubMed          Journal:  Am J Reprod Immunol        ISSN: 1046-7408            Impact factor:   3.886


  3 in total

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Authors:  Sook-Eun Park; Marcus Lawson; Robert Dantzer; Keith W Kelley; Robert H McCusker
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Authors:  Alexander Lee; Navya Kanuri; Yuanhao Zhang; Gregory S Sayuk; Ellen Li; Matthew A Ciorba
Journal:  PLoS One       Date:  2014-12-26       Impact factor: 3.240

  3 in total

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