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Literature DB >> 2015118

St Cyres lecture. Endothelium in control.

A H Henderson¹.

Abstract

Mesh：

Substances：
Nitric Oxide

Year: 1991 PMID： 2015118 PMCID： PMC1024532 DOI： 10.1136/hrt.65.3.116

Source DB: PubMed Journal: Br Heart J ISSN： 0007-0769

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159 in total

Review 1. The endocardium.

Authors: D L Brutsaert
Journal: Annu Rev Physiol Date: 1989 Impact factor: 19.318

Review 2. Endothelium-derived relaxing factor.

Authors: J A Angus; T M Cocks
Journal: Pharmacol Ther Date: 1989 Impact factor: 12.310

3. The role of EDRF in flow distribution: a microangiographic study of the rabbit isolated ear.

Authors: T M Griffith; D H Edwards; R L Davies; A H Henderson
Journal: Microvasc Res Date: 1989-03 Impact factor: 3.514

4. Dietary omega 3 fatty acids and endothelium-dependent relaxations in porcine coronary arteries.

Authors: H Shimokawa; P M Vanhoutte
Journal: Am J Physiol Date: 1989-04

5. Nitric oxide-generating vasodilators and 8-bromo-cyclic guanosine monophosphate inhibit mitogenesis and proliferation of cultured rat vascular smooth muscle cells.

Authors: U C Garg; A Hassid
Journal: J Clin Invest Date: 1989-05 Impact factor: 14.808

6. Macrophage oxidation of L-arginine to nitrite and nitrate: nitric oxide is an intermediate.

Authors: M A Marletta; P S Yoon; R Iyengar; C D Leaf; J S Wishnok
Journal: Biochemistry Date: 1988-11-29 Impact factor: 3.162

7. Influence of experimental diabetes on the mechanical responses of canine coronary arteries: role of endothelium.

Authors: D Gebremedhin; M Z Koltai; G Pogátsa; K Magyar; P Hadházy
Journal: Cardiovasc Res Date: 1988-08 Impact factor: 10.787

8. Human red blood cells inhibit endothelium-derived relaxing factor (EDRF) activity.

Authors: H G Evans; H C Ryley; I Hallett; M J Lewis
Journal: Eur J Pharmacol Date: 1989-04-25 Impact factor: 4.432

9. Role of endothelium-derived nitric oxide in the regulation of blood pressure.

Authors: D D Rees; R M Palmer; S Moncada
Journal: Proc Natl Acad Sci U S A Date: 1989-05 Impact factor: 11.205

10. Atrial natriuretic factor inhibits proliferation of vascular smooth muscle cells stimulated by platelet-derived growth factor.

Authors: T J Abell; A M Richards; H Ikram; E A Espiner; T Yandle
Journal: Biochem Biophys Res Commun Date: 1989-05-15 Impact factor: 3.575

25 in total

St Cyres lecture. Endothelium in control.

Review 1. The endocardium.

Review 2. Endothelium-derived relaxing factor.

3. The role of EDRF in flow distribution: a microangiographic study of the rabbit isolated ear.

4. Dietary omega 3 fatty acids and endothelium-dependent relaxations in porcine coronary arteries.

5. Nitric oxide-generating vasodilators and 8-bromo-cyclic guanosine monophosphate inhibit mitogenesis and proliferation of cultured rat vascular smooth muscle cells.

6. Macrophage oxidation of L-arginine to nitrite and nitrate: nitric oxide is an intermediate.

7. Influence of experimental diabetes on the mechanical responses of canine coronary arteries: role of endothelium.

8. Human red blood cells inhibit endothelium-derived relaxing factor (EDRF) activity.

9. Role of endothelium-derived nitric oxide in the regulation of blood pressure.

10. Atrial natriuretic factor inhibits proliferation of vascular smooth muscle cells stimulated by platelet-derived growth factor.

1. Reperfusion Injury: Does It Exist and Does It Have Clinical Relevance?

2. The effect of testosterone on regional blood flow in prepubertal anaesthetized pigs.

3. Angioplasty and restenosis.

Review 4. Delivery and monitoring of inhaled nitric oxide.

Review 5. The role of exercise training in chronic heart failure.

6. Vascular endothelium: in defense of blood fluidity.

7. Induction of a calcium-independent NO synthase by hypercholesterolaemia in the rabbit.

8. Porcine ventricular endocardial cells in culture express the inducible form of nitric oxide synthase.

9. Non-invasive measurement of human endothelium dependent arterial responses: accuracy and reproducibility.

10. Apolipoprotein e4 allele and endothelium-dependent arterial dilation in Type 2 diabetes mellitus without angiopathy.