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17 in total

1. Microglial activation by Alzheimer amyloid precursor protein and modulation by apolipoprotein E.

Authors: S W Barger; A D Harmon
Journal: Nature Date: 1997-08-28 Impact factor: 49.962

2. APP locus duplication causes autosomal dominant early-onset Alzheimer disease with cerebral amyloid angiopathy.

Authors: Anne Rovelet-Lecrux; Didier Hannequin; Gregory Raux; Nathalie Le Meur; Annie Laquerrière; Anne Vital; Cécile Dumanchin; Sébastien Feuillette; Alexis Brice; Martine Vercelletto; Frédéric Dubas; Thierry Frebourg; Dominique Campion
Journal: Nat Genet Date: 2005-12-20 Impact factor: 38.330

3. Promoter mutations that increase amyloid precursor-protein expression are associated with Alzheimer disease.

Authors: Jessie Theuns; Nathalie Brouwers; Sebastiaan Engelborghs; Kristel Sleegers; Veerle Bogaerts; Ellen Corsmit; Tim De Pooter; Cornelia M van Duijn; Peter P De Deyn; Christine Van Broeckhoven
Journal: Am J Hum Genet Date: 2006-04-10 Impact factor: 11.025

4. Neuronal-glial interactions mediated by interleukin-1 enhance neuronal acetylcholinesterase activity and mRNA expression.

Authors: Y Li; L Liu; J Kang; J G Sheng; S W Barger; R E Mrak; W S Griffin
Journal: J Neurosci Date: 2000-01-01 Impact factor: 6.167

Review 5. Glia and their cytokines in progression of neurodegeneration.

Authors: Robert E Mrak; W Sue T Griffin
Journal: Neurobiol Aging Date: 2005-03 Impact factor: 4.673

6. App gene dosage modulates endosomal abnormalities of Alzheimer's disease in a segmental trisomy 16 mouse model of down syndrome.

Authors: Anne M Cataldo; Suzana Petanceska; Corrinne M Peterhoff; Nicole B Terio; Charles J Epstein; Angela Villar; Elaine J Carlson; Matthias Staufenbiel; Ralph A Nixon
Journal: J Neurosci Date: 2003-07-30 Impact factor: 6.167

Excess betaCTF, not Abeta: the culprit in Alzheimer-related endocytic dysfunction.

1. Microglial activation by Alzheimer amyloid precursor protein and modulation by apolipoprotein E.

2. APP locus duplication causes autosomal dominant early-onset Alzheimer disease with cerebral amyloid angiopathy.

3. Promoter mutations that increase amyloid precursor-protein expression are associated with Alzheimer disease.

4. Neuronal-glial interactions mediated by interleukin-1 enhance neuronal acetylcholinesterase activity and mRNA expression.

Review 5. Glia and their cytokines in progression of neurodegeneration.

6. App gene dosage modulates endosomal abnormalities of Alzheimer's disease in a segmental trisomy 16 mouse model of down syndrome.

7. Alzheimer's disease in Down's syndrome: clinicopathologic studies.

8. Occurrence of neuropathological changes and dementia of Alzheimer's disease in Down's syndrome.

9. Protease inhibitor domain encoded by an amyloid protein precursor mRNA associated with Alzheimer's disease.

10. Brain interleukin 1 and S-100 immunoreactivity are elevated in Down syndrome and Alzheimer disease.

1. Neuroinflammatory Cytokines-The Common Thread in Alzheimer's Pathogenesis.