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7 in total

1. The leukemogenicity of AML1-ETO is dependent on site-specific lysine acetylation.

Authors: Lan Wang; Alexander Gural; Xiao-Jian Sun; Xinyang Zhao; Fabiana Perna; Gang Huang; Megan A Hatlen; Ly Vu; Fan Liu; Haiming Xu; Takashi Asai; Hao Xu; Tony Deblasio; Silvia Menendez; Francesca Voza; Yanwen Jiang; Philip A Cole; Jinsong Zhang; Ari Melnick; Robert G Roeder; Stephen D Nimer
Journal: Science Date: 2011-07-14 Impact factor: 47.728

2. A genome-wide aberrant RNA splicing in patients with acute myeloid leukemia identifies novel potential disease markers and therapeutic targets.

Authors: Sophia Adamia; Benjamin Haibe-Kains; Patrick M Pilarski; Michal Bar-Natan; Samuel Pevzner; Herve Avet-Loiseau; Laurence Lode; Sigitas Verselis; Edward A Fox; John Burke; Ilene Galinsky; Ibiayi Dagogo-Jack; Martha Wadleigh; David P Steensma; Gabriela Motyckova; Daniel J Deangelo; John Quackenbush; Richard Stone; James D Griffin
Journal: Clin Cancer Res Date: 2013-11-27 Impact factor: 12.531

3. NOTCH2 and FLT3 gene mis-splicings are common events in patients with acute myeloid leukemia (AML): new potential targets in AML.

Authors: Sophia Adamia; Michal Bar-Natan; Benjamin Haibe-Kains; Patrick M Pilarski; Christian Bach; Samuel Pevzner; Teresa Calimeri; Herve Avet-Loiseau; Laurence Lode; Sigitas Verselis; Edward A Fox; Ilene Galinsky; Steven Mathews; Ibiayi Dagogo-Jack; Martha Wadleigh; David P Steensma; Gabriela Motyckova; Daniel J Deangelo; John Quackenbush; Daniel G Tenen; Richard M Stone; James D Griffin
Journal: Blood Date: 2014-02-26 Impact factor: 22.113

A novel exon in AML1-ETO negatively influences the clonogenic potential of the t(8;21) in acute myeloid leukemia.

1. The leukemogenicity of AML1-ETO is dependent on site-specific lysine acetylation.

2. A genome-wide aberrant RNA splicing in patients with acute myeloid leukemia identifies novel potential disease markers and therapeutic targets.

3. NOTCH2 and FLT3 gene mis-splicings are common events in patients with acute myeloid leukemia (AML): new potential targets in AML.

4. RNA-Seq analysis identifies aberrant RNA splicing of TRIP12 in acute myeloid leukemia patients at remission.

5. Acute myeloid leukemia with the t(8;21) translocation: clinical consequences and biological implications.

Review 6. Fusion transcripts: Unexploited vulnerabilities in cancer?

Review 7. AML1/ETO and its function as a regulator of gene transcription via epigenetic mechanisms.