Literature DB >> 20095175

Early treatment with nebulised salbutamol worsens physiological measures and does not improve survival following phosgene induced acute lung injury.

C Grainge1, R Brown, B J Jugg, A J Smith, T M Mann, J Jenner, P Rice, D A Parkhouse.   

Abstract

OBJECTIVES: To examine the effectiveness of nebulised salbutamol in the treatment of phosgene induced acute lung injury.
METHOD: Using previously validated methods, 12 anaesthetised large white pigs were exposed to phosgene (Ct 1978 +/- 8 mg min m(-3)), established on mechanical ventilation and randomised to treatment with either nebulised salbutamol (2.5 mg per dose) or saline control. Treatments were given 1, 5, 9, 13, 17 and 21 hours following phosgene exposure. The animals were followed to 24 hours following phosgene exposure.
RESULTS: Salbutamol treatment had no effect on mortality and had a deleterious effect on arterial oxygenation, shunt fraction and heart rate. There was a reduction in the number of neutrophils from 24.0% +/- 4.4 to 12.17% +/- 2.1 (p < 0.05) in bronchoalveolar lavage, with some small decreases in inflammatory mediators in bronchoalveolar lavage but not in plasma.
CONCLUSION: Nebulised salbutamol treatment following phosgene induced acute lung injury does not improve survival, and worsens various physiological parameters including arterial oxygen partial pressure and shunt fraction. Salbutamol treatment reduces neutrophil influx into the lung. Its sole use following phosgene exposure is not recommended.

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Year:  2009        PMID: 20095175     DOI: 10.1136/jramc-155-02-05

Source DB:  PubMed          Journal:  J R Army Med Corps        ISSN: 0035-8665            Impact factor:   1.285


  6 in total

Review 1.  The injured lung: clinical issues and experimental models.

Authors:  B J A Jugg; A J Smith; S J Rudall; P Rice
Journal:  Philos Trans R Soc Lond B Biol Sci       Date:  2011-01-27       Impact factor: 6.237

2.  An Official American Thoracic Society Workshop Report: Chemical Inhalational Disasters. Biology of Lung Injury, Development of Novel Therapeutics, and Medical Preparedness.

Authors:  Eleanor M Summerhill; Gary W Hoyle; Sven-Eric Jordt; Bronwen J Jugg; James G Martin; Sadis Matalon; Steven E Patterson; David J Prezant; Alfred M Sciuto; Erik R Svendsen; Carl W White; Livia A Veress
Journal:  Ann Am Thorac Soc       Date:  2017-06

3.  Salvianolic acid B attenuates lipopolysaccharide-induced acute lung injury in rats through inhibition of apoptosis, oxidative stress and inflammation.

Authors:  Da-Hai Zhao; Yu-Jie Wu; Shu-Ting Liu; Rong-Yu Liu
Journal:  Exp Ther Med       Date:  2017-06-01       Impact factor: 2.447

4.  Phosgene exposure: a case of accidental industrial exposure.

Authors:  Lewis S Hardison; Edward Wright; Anthony F Pizon
Journal:  J Med Toxicol       Date:  2014-03

Review 5.  Phosgene-induced acute lung injury (ALI): differences from chlorine-induced ALI and attempts to translate toxicology to clinical medicine.

Authors:  Wenli Li; Juergen Pauluhn
Journal:  Clin Transl Med       Date:  2017-06-02

6.  Phosgene-Induced acute lung injury: Approaches for mechanism-based treatment strategies.

Authors:  Chao Cao; Lin Zhang; Jie Shen
Journal:  Front Immunol       Date:  2022-08-02       Impact factor: 8.786

  6 in total

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