Literature DB >> 20035620

Overexpression of beta-arrestin 2 in peripheral blood mononuclear cells of patients with cryptococcal meningitis.

Rong Xia1, Zhide Hu, Yi Sun, Sunxiao Chen, Mingli Gu, Ye Zhou, Zhijun Han, Renqian Zhong, Anmei Deng, Hai Wen.   

Abstract

Cryptococcal meningitis is often associated with elevated IL-10 levels, which suggest a dysregulation in the antifungal immune response. beta-Arrestin 2 plays a major role in desensitization of G-protein-coupled receptors involved in the immune responses, provides a scaffolding platform for modification of many signal transduction proteins, and binds Src and MAP kinases family members. This study compared the levels of beta-arrestin 2 mRNA and protein in peripheral blood mononuclear cells (PBMC) of patients with cryptococcal meningitis detected. The interferon-gamma (IFN-gamma) serum concentration was determined with enzyme-linked immunosorbent assay (ELISA) to reveal its relationship with beta-arrestin 2. The effect of modulation of beta-arrestin 2 on cytotoxic activity against Cryptococcus was explored via transfection and interference of beta-arrestin 2. PBMCs of patients with cryptococcal meningitis exhibited significantly elevated levels of beta-arrestin 2 and a positive correlation between beta-arrestin 2 and IL-10 levels existed in patients' serum, but a negative correlation was found between beta-arrestin 2 and IFN-gamma expression. In conclusion, elevated expression of beta-arrestin 2 in PBMCs of patients with cryptococcal meningitis correlated with a reduced cytotoxic activity against Cryptococcus. This study suggests that reduced beta-arrestin 2 mRNA levels or inhibition of beta-arrestin 2 activity may augment INF-gamma production, and ultimately, the anti-Cryptococcus immune response of infected patients.

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Year:  2010        PMID: 20035620     DOI: 10.1089/jir.2009.0017

Source DB:  PubMed          Journal:  J Interferon Cytokine Res        ISSN: 1079-9907            Impact factor:   2.607


  7 in total

1.  TLR4-dependent internalization of CX3CR1 aggravates sepsis-induced immunoparalysis.

Authors:  Xin-Yu Ge; Shang-Ping Fang; Miao Zhou; Jing Luo; Juan Wei; Xue-Ping Wen; Xiao-Di Yan; Zui Zou
Journal:  Am J Transl Res       Date:  2016-12-15       Impact factor: 4.060

2.  Dopamine D2 receptor restricts astrocytic NLRP3 inflammasome activation via enhancing the interaction of β-arrestin2 and NLRP3.

Authors:  Jialei Zhu; Zhaoli Hu; Xiaojuan Han; Dongshuo Wang; Qingling Jiang; Jianhua Ding; Ming Xiao; Cong Wang; Ming Lu; Gang Hu
Journal:  Cell Death Differ       Date:  2018-05-21       Impact factor: 15.828

3.  β-Arrestin 2 negatively regulates Toll-like receptor 4 (TLR4)-triggered inflammatory signaling via targeting p38 MAPK and interleukin 10.

Authors:  Hui Li; Dan Hu; Huimin Fan; Ying Zhang; Gene D LeSage; Yi Caudle; Charles Stuart; Zhongmin Liu; Deling Yin
Journal:  J Biol Chem       Date:  2014-07-10       Impact factor: 5.157

Review 4.  β-Arrestins in the immune system.

Authors:  Dianhua Jiang; Ting Xie; Jiurong Liang; Paul W Noble
Journal:  Prog Mol Biol Transl Sci       Date:  2013       Impact factor: 3.622

5.  Systematic expression profiling analysis identifies specific microRNA-gene interactions that may differentiate between active and latent tuberculosis infection.

Authors:  Lawrence Shih-Hsin Wu; Shih-Wei Lee; Kai-Yao Huang; Tzong-Yi Lee; Paul Wei-Che Hsu; Julia Tzu-Ya Weng
Journal:  Biomed Res Int       Date:  2014-09-04       Impact factor: 3.411

Review 6.  Multifaceted role of β-arrestins in inflammation and disease.

Authors:  D Sharma; N Parameswaran
Journal:  Genes Immun       Date:  2015-09-17       Impact factor: 2.676

7.  β-arrestin2 functions as a key regulator in the sympathetic-triggered immunodepression after stroke.

Authors:  Huan Wang; Qi-Wen Deng; Ai-Ni Peng; Fang-Lan Xing; Lei Zuo; Shuo Li; Zheng-Tian Gu; Fu-Ling Yan
Journal:  J Neuroinflammation       Date:  2018-04-10       Impact factor: 8.322

  7 in total

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