Literature DB >> 19996067

Progress toward identifying potential markers for preeclampsia: role of agonistic autoantibody to the angiotensin II type I receptor.

Babbette LaMarca.   

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Year:  2009        PMID: 19996067      PMCID: PMC3061341          DOI: 10.1161/HYPERTENSIONAHA.109.141465

Source DB:  PubMed          Journal:  Hypertension        ISSN: 0194-911X            Impact factor:   10.190


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  14 in total

1.  Antibodies from preeclamptic patients stimulate increased intracellular Ca2+ mobilization through angiotensin receptor activation.

Authors:  Theingi M Thway; Sergiy G Shlykov; Mary-Clare Day; Barbara M Sanborn; Larry C Gilstrap; Yang Xia; Rodney E Kellems
Journal:  Circulation       Date:  2004-09-21       Impact factor: 29.690

Review 2.  Agonistic antibodies directed at the angiotensin II, AT1 receptor in preeclampsia.

Authors:  Ralf Dechend; Volker Homuth; Gerd Wallukat; Dominik N Müller; Manja Krause; Joachim Dudenhausen; Hermann Haller; Friedrich C Luft
Journal:  J Soc Gynecol Investig       Date:  2006-02

3.  Maternal autoantibodies from preeclamptic patients activate angiotensin receptors on human mesangial cells and induce interleukin-6 and plasminogen activator inhibitor-1 secretion.

Authors:  Sol M Bobst; Mary-Clare Day; Larry C Gilstrap; Yang Xia; Rodney E Kellems
Journal:  Am J Hypertens       Date:  2005-03       Impact factor: 2.689

4.  Relation between circulating angiotensin II type 1 receptor agonistic autoantibodies and soluble fms-like tyrosine kinase 1 in the pathogenesis of preeclampsia.

Authors:  H Stepan; R Faber; N Wessel; G Wallukat; H-P Schultheiss; T Walther
Journal:  J Clin Endocrinol Metab       Date:  2006-03-28       Impact factor: 5.958

5.  Patients with preeclampsia develop agonistic autoantibodies against the angiotensin AT1 receptor.

Authors:  G Wallukat; V Homuth; T Fischer; C Lindschau; B Horstkamp; A Jüpner; E Baur; E Nissen; K Vetter; D Neichel; J W Dudenhausen; H Haller; F C Luft
Journal:  J Clin Invest       Date:  1999-04       Impact factor: 14.808

6.  AT1 receptor agonistic antibodies from preeclamptic patients stimulate NADPH oxidase.

Authors:  Ralf Dechend; Christiane Viedt; Dominik N Müller; Bernhard Ugele; Ralf P Brandes; Gerd Wallukat; Joon-Keun Park; Jurgen Janke; Peter Barta; Jurgen Theuer; Anette Fiebeler; Volker Homuth; Rainer Dietz; Hermann Haller; Jörg Kreuzer; Friedrich C Luft
Journal:  Circulation       Date:  2003-02-24       Impact factor: 29.690

7.  Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia.

Authors:  Sharon E Maynard; Jiang-Yong Min; Jaime Merchan; Kee-Hak Lim; Jianyi Li; Susanta Mondal; Towia A Libermann; James P Morgan; Frank W Sellke; Isaac E Stillman; Franklin H Epstein; Vikas P Sukhatme; S Ananth Karumanchi
Journal:  J Clin Invest       Date:  2003-03       Impact factor: 14.808

8.  Hypertension produced by reduced uterine perfusion in pregnant rats is associated with increased soluble fms-like tyrosine kinase-1 expression.

Authors:  Jeffrey S Gilbert; Sara A Babcock; Joey P Granger
Journal:  Hypertension       Date:  2007-10-08       Impact factor: 10.190

Review 9.  Recent Insights into the pathogenesis of pre-eclampsia.

Authors:  J M Roberts; K Y Lain
Journal:  Placenta       Date:  2002-05       Impact factor: 3.481

10.  Autoantibody from women with preeclampsia induces soluble Fms-like tyrosine kinase-1 production via angiotensin type 1 receptor and calcineurin/nuclear factor of activated T-cells signaling.

Authors:  Cissy Chenyi Zhou; Shakil Ahmad; Tiejuan Mi; Shahrzad Abbasi; Lingwei Xia; Mary-Clare Day; Susan M Ramin; Asif Ahmed; Rodney E Kellems; Yang Xia
Journal:  Hypertension       Date:  2008-02-07       Impact factor: 10.190

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  3 in total

1.  IL-6-induced pathophysiology during pre-eclampsia: potential therapeutic role for magnesium sulfate?

Authors:  Babbette Lamarca; Justin Brewer; Kedra Wallace
Journal:  Int J Interferon Cytokine Mediat Res       Date:  2011-07-01

2.  Maternal plasma fetuin-A concentration is lower in patients who subsequently developed preterm preeclampsia than in uncomplicated pregnancy: a longitudinal study.

Authors:  Piya Chaemsaithong; Roberto Romero; Adi L Tarca; Steven J Korzeniewski; Alyse G Schwartz; Jezid Miranda; Ahmed I Ahmed; Zhong Dong; Sonia S Hassan; Lami Yeo; Tinnakorn Tinnakorn
Journal:  J Matern Fetal Neonatal Med       Date:  2014-09-29

3.  Autoantibody-mediated complement C3a receptor activation contributes to the pathogenesis of preeclampsia.

Authors:  Wei Wang; Roxanna A Irani; Yujin Zhang; Susan M Ramin; Sean C Blackwell; Lijian Tao; Rodney E Kellems; Yang Xia
Journal:  Hypertension       Date:  2012-08-06       Impact factor: 10.190

  3 in total

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