Literature DB >> 19940479

No evidence of PGRN or MAPT gene dosage alterations in a collection of patients with frontotemporal lobar degeneration.

Lena Skoglund1, Sofie Ingvast, Toshifumi Matsui, Stefanie H Freeman, Matthew P Frosch, Rosemarie Brundin, Vilmantas Giedraitis, John H Growdon, Bradley T Hyman, Lars Lannfelt, Martin Ingelsson, Anna Glaser.   

Abstract

BACKGROUND/AIMS: Alterations in gene dosage have recently been associated with neurodegenerative disorders, such as Alzheimer's disease and Parkinson's disease, and deletions of the progranulin (PGRN) locus were recently described in patients with frontotemporal lobar degeneration (FTLD). FTLD is a genetically complex neurodegenerative disorder with mutations in the PGRN and the microtubule-associated protein tau (MAPT) genes being the most common known causes of familial FTLD. In this study, we investigated 39 patients with FTLD, previously found negative for mutations in PGRN and MAPT, for copy number alterations of these 2 genes.
METHODS: Gene dosage analysis of PGRN and MAPT was performed using multiplex ligation-dependent probe amplification.
RESULTS: We did not identify any PGRN or MAPT gene dosage variations in the 39 FTLD patients investigated.
CONCLUSION: We therefore conclude that alterations in gene copy number of PGRN and MAPT are not a cause of disease in this collection of FTLD patients. Copyright 2009 S. Karger AG, Basel.

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Year:  2009        PMID: 19940479      PMCID: PMC7077072          DOI: 10.1159/000260046

Source DB:  PubMed          Journal:  Dement Geriatr Cogn Disord        ISSN: 1420-8008            Impact factor:   2.959


  38 in total

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Journal:  Brain       Date:  2008-01-29       Impact factor: 13.501

10.  The prevalence of frontotemporal dementia.

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