Literature DB >> 19934107

Tumour necrosis factor {alpha} blockade reduces circulating N-terminal pro-brain natriuretic peptide levels in patients with active rheumatoid arthritis: results from a prospective cohort study.

Mike J L Peters1, Paul Welsh, Iain B McInnes, Gertjan Wolbink, Ben A C Dijkmans, Naveed Sattar, Michael T Nurmohamed.   

Abstract

BACKGROUND: Patients with rheumatoid arthritis (RA) are at increased risk of heart failure and vascular events. Small increases in circulating N-terminal pro-brain natriuretic peptide (NT-proBNP) are associated with an increased risk of a cardiovascular event, and high levels signal left ventricular dysfunction. Data on the effects of tumour necrosis factor alpha(TNFalpha) blocking agents on circulating NT-proBNP levels in patients with active RA are lacking but may be informative.
METHODS: 171 consecutive patients with RA (28-joint disease activity score >3.2) without congestive heart failure (NYHA class III or IV) were scheduled to receive adalimumab once every 2 weeks. Serum NT-proBNP concentrations were measured simultaneously on stored baseline and 16-week samples. Paired sample t tests were used to observe differences in biomarkers before and after adalimumab administration. Correlations between the biomarkers and changes in circulating log NT-proBNP levels were evaluated with the Pearson test and multivariable linear regression analyses of correlates were performed (forward selection procedure).
RESULTS: Circulating levels of NT-proBNP decreased significantly after 16 weeks of adalimumab administration (median NT-proBNP 83.0 pg/ml vs 69.5 pg/ml, p=0.004). Changes in NT-proBNP levels were associated with changes in pulse pressure (r=0.18, p=0.02), systolic blood pressure (r=0.16, p=0.04) and erythrocyte sedimentation rate (r=0.18, p=0.02). On multivariable analysis, changes in pulse pressure and erythrocyte sedimentation rate remained independently associated with changes in circulating NT-proBNP levels.
CONCLUSIONS: These observations show that blocking TNFalpha in patients with RA without evident heart failure decreases NT-proBNP levels by about 18%. This suggests no treatment-induced deterioration in cardiac function and a potential cardiovascular risk benefit.

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Year:  2009        PMID: 19934107     DOI: 10.1136/ard.2009.119412

Source DB:  PubMed          Journal:  Ann Rheum Dis        ISSN: 0003-4967            Impact factor:   19.103


  16 in total

1.  NT-proBNP levels may be influenced by inflammation in active ankylosing spondylitis receiving TNF blockers: a pilot study.

Authors:  Julio C B Moraes; Ana C M Ribeiro; Carla G S Saad; Alessandro C Lianza; Clovis A Silva; Eloísa Bonfá
Journal:  Clin Rheumatol       Date:  2013-02-05       Impact factor: 2.980

2.  Amino-terminal pro-brain natriuretic peptide as a prognostic marker in patients with rheumatoid arthritis.

Authors:  Bożena Targońska-Stępniak; Maria Majdan
Journal:  Clin Rheumatol       Date:  2010-11-26       Impact factor: 2.980

3.  Acute and long-term effect of infliximab on humoral and echocardiographic parameters in patients with chronic inflammatory diseases.

Authors:  L'ubomír Tomáš; Ivica Lazúrová; Lýdia Pundová; Mária Oetterová; Mária Zakuciová; Darina Petrášová; Martin Studenčan
Journal:  Clin Rheumatol       Date:  2012-09-26       Impact factor: 2.980

Review 4.  Cardiac Impairment in Rheumatoid Arthritis and Influence of Anti-TNFα Treatment.

Authors:  Ivica Lazúrová; Ľubomír Tomáš
Journal:  Clin Rev Allergy Immunol       Date:  2017-06       Impact factor: 8.667

5.  Increased extracellular water measured by bioimpedance and by increased serum levels of atrial natriuretic peptide in RA patients-signs of volume overload.

Authors:  Rainer H Straub; Boris Ehrenstein; Florian Günther; Luise Rauch; Nadezhda Trendafilova; Dario Boschiero; Joachim Grifka; Martin Fleck
Journal:  Clin Rheumatol       Date:  2016-04-26       Impact factor: 2.980

6.  [Rheumatoid arthritis. Systemic inflammation and cardiovascular morbidity].

Authors:  E Decker; U Müller-Ladner
Journal:  Internist (Berl)       Date:  2013-04       Impact factor: 0.743

7.  NH2-terminal probrain natriuretic peptide is associated with diabetes complications in the EURODIAB Prospective Complications Study: the role of tumor necrosis factor-α.

Authors:  Gabriella Gruden; Federica Barutta; Nish Chaturvedi; Casper Schalkwijk; Coen D Stehouwer; Silvia Pinach; Maria Manzo; Maria Loiacono; Marinella Tricarico; Giulio Mengozzi; Daniel R Witte; John H Fuller; Paolo Cavallo Perin; Graziella Bruno
Journal:  Diabetes Care       Date:  2012-06-14       Impact factor: 19.112

Review 8.  Evolutionary medicine and chronic inflammatory state--known and new concepts in pathophysiology.

Authors:  Rainer H Straub
Journal:  J Mol Med (Berl)       Date:  2012-01-22       Impact factor: 4.599

9.  N-terminal pro-brain-type natriuretic peptide (NT-pro-BNP) and mortality risk in early inflammatory polyarthritis: results from the Norfolk Arthritis Registry (NOAR).

Authors:  Hoda Mirjafari; Paul Welsh; Suzanne M M Verstappen; Paddy Wilson; Tarnya Marshall; Helena Edlin; Diane Bunn; Jacqueline Chipping; Mark Lunt; Deborah P M Symmons; Naveed Sattar; Ian N Bruce
Journal:  Ann Rheum Dis       Date:  2013-03-19       Impact factor: 19.103

10.  Effect of IL-6 receptor blockade on high-sensitivity troponin T and NT-proBNP in rheumatoid arthritis.

Authors:  Paul Welsh; Katie Tuckwell; Iain B McInnes; Naveed Sattar
Journal:  Atherosclerosis       Date:  2016-10-08       Impact factor: 5.162

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